I. BLEEDING IN PREGNANCY
Table 9. Outline of Classification
I. First Trimester Bleeding
A. Abortion
1. Spontaneous
a. Threatened
b. Imminent
c. Complete
d. Incomplete
2. Induced
a. Therapeutic
b. Illegal
B. Ectopic Pregnancy
1. Tubal – most common
2. Cervical
3. Ovarian
4. Abdominal
II. Second Trimester Bleeding
A. Hydatidiform Mole
B. Incompetent Cervical Os
III. Third Trimester Bleeding
A. Placenta previa
B. Abruptio placenta
A. ABORTION – any interruption in pregnancy before the age of viability.
1. Spontaneous – occurs from natural causes, blighted ovum/germ plasma defect (most common cause – it is nature’s way of eliminating the birth of a congenitally defective baby); implantation or hormonal abnormality; following trauma, infection (e.g., rubella, influenza) or emotional problems
a. Threatened
• Symptom: bright red vaginal bleeding which is moderate in amount
• Management:
• Complete bed rest for 24-48 hours; if bleeding will stop it usually steps within this time
• Coitus is restricted for 2 weeks after bleeding has stopped in order to prevent further bleeding or infection
• Endocrine/hormonal therapy
• Advise patient to save all pads, clots and expelled tissues
b. Imminent/inevitable
• Symptom: Bright red vaginal bleeding which is moderate in amount and accompanied by uterine contractions and cervical dilatation. Loss of the products of conception is inevitable.
• Management: depends on whether it is
• Complete abortion – all products of conception are expelled; bleeding is minimal and self-limiting. No intervention is therefore needed.
• Incomplete abortion – part of the conceptus, usually the fetus, is expelled, but membranes or placental fragments are retained. D & C is indicated as management.
c. Missed abortion – fetus dies in utero but is not expelled. Usually discovered at a prenatal visit when fundic height is measure and no increase is demonstrated or when previously heard fetal heart tones are no longer present. In 2 weeks’ time, signs of abortion should occur; otherwise, labor will have to be induced to prevent hypofibrinoginemia or sepsis.
2. Induced – is never allowed in the Philippines
a. Therapeutic – performed by a doctor in a controlled hospital or clinic setting for a medical or a legal reason. Also known as medical, planned or legal abortion.
b. Illegal
B. ECTOPIC PREGNANCY – any gestation located outside the uterine cavity.
1. Signs and symptoms – since the wall of the Fallopian tube is not sufficiently elastic, it ruptures within the first 12 weeks of gestation as it can no longer give way for the growing fetus:
a. peri-umbilical colicky pain which mimics appendicitis (in bleeding wherein there is no exit or egress of blood from the body, pain is the outstanding symptom; this pain differentiates ectopic pregnancy from abortion).
b. may radiate to shoulder & neck if internal bleeding reach level of diaphragm
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d. Excruciating pain when cervix is moved on IE
e. Signs of shock: falling BP, tachycardia, lightheadedness
• Ruptured ectopic pregnancy is an emergency situation.
2. Diagnosis:
a. Pelvic exam-reveal adnexae or cul de sac mass/cul de sac of Douglas
b. Culdocentesis-aspirate fluid
c. Culdotomy
d. Laparoscopy
e. D&C
3. Management:
a. Laparotomy – if Fallopian tube can still be repaired and preserved, but the pregnancy has to be terminated
b. Salpingectomy + blood transfusion
c. Salphingo-oopherectomy
3. Nursing care – combat shock:
a. Elevate foot of the bed
b. Maintain body heat by hot water bottles and blankets
C. HYDATIDIFORM MOLE – developmental anomaly of the placenta resulting in proliferation and degeneration of the chorionic villi
1. Incidence: it is the most common lesion anteceding choriocarcinoma. It occurs most often in women:
a. From low socioeconomic backgrounds with low protein intake
b. Over 35 years and under 18 years of age
2. Signs and symptoms:
a. Because of rapid proliferation of placental tissues and, therefore, high levels of HCG:
• Highly positive urine test for pregnancy (that is why a positive pregnancy test cannot be considered a positive sign of pregnancy)
• Nausea and vomiting is usually marked
• Rapid increase in fundic height
b. Toxemia signs and symptoms appear before the 24th week of gestation
c. No fetal heart tones
d. Vaginal bleeding seen as clear, fluid-filled grape-sized vesicles
4. Management:
a. D & C to evacuate the mole
b. Prophylactic course of Methotrexate, the drug of choice for choriocarcinoma
c. Urine testing for one year to find out if new villi are developing. Contraceptives (but not the pills) have to used to as not to confuse the results.
D. INCOMPETENT CERVICAL OS – cervix dilates prematurely. It is the chief cause of habitual abortion (=3 or more consecutive abortions)
1. Causes:
a. Congenital developmental factors
b. Endocrine factors
c. Trauma to the cervix
2. Signs and symptoms:
a. Presence of show and uterine contractions
b. Rupture of membrane
c. Painless cervical dilatation
3. Management: McDonal/Shirodkar-Barter procedure – a cerclage procedure wherein purse-string sutures are place around the cervix on the 14th-18th week of gestation. These are removed during vaginal delivery (if McDonald’s method, since sutures are temporary), or the patient delivers by Cesarean section (if Shirodkar method, since sutures are permanent).
E. PLACENTA PREVIA – low implantation of the placenta so that it is in the way of the presenting part.
1. Predisposing factors:
a. Increasing parity
b. Advanced maternal age
c. Rapid succession of pregnancies
2. Types:
a. Low lying
b. Partial
c. Complete
3. Diagnosis – made by means of symptoms and ultrasound – also known as Ultrasonic Echo Sounding or Sonar, uses intermittent waves of very high frequency (above audible range) to “picture the fetus”. Sound waves are projected towards the mother’s abdomen, are reflected back and converted into electrical impulses and recorded on a permanent graph paper.
a. Preparation:
• Explain the procedure to the patient, informing her that it is painless and there are no known ill effects
• Empty the bladder BUT ask the patient to take 6 glasses of water afterwards in order to dilate the bladder. A full bladder displaces the bowel and, therefore, permits better visualization of the pelvis and its contents.
b. Clinical uses:
• Diagnose pregnancy as early as 5-6 weeks gestational age
• Can establish that the fetus is increasing in size and, therefore, can predict EDC
• Can determine gestational age by measuring the biparietal diameter of the fetal skill (if it is more than 8.5 cm, it more or less weighs more than 2500 gms); therefore, can diagnose intrauterine growth retardation, hydrocephaly, microcephaly and anencephaly
• Can demonstrate size and growth rate of the amniotic sac; therefore, can identify polyhydramnic, oligohydramnios
• Can confirm presence, size and location of the placenta; therefore, is valuable in diagnosing previa and H-mole
• Can diagnose multiple pregnancy
• Can visualize ascites, polycystic kidneys, ovarian cysts, etc.
• Can determine baby’s sex (during third trimester and if in cephalic presentation)
4. First and most constant symptom: painless bright red vaginal bleeding due to tearing of placental attachment as a consequence of the dilatation of the internal os
5. Management:
a. Complete bed rest
b. Monitor vital signs of the mother and the fetal heart rate
c. Prepare oxygen and blood
d. No attempt is made at doing internal exam. If ever it is to be done, it is done in a double set-up (done in the operating room wherein the patient has already signed the consent form, preop medications have been given, abdominal prep has been done, etc., so that if the placenta is accidentally detached because of the IE, CS can be done immediately).
6. Complications:
a. Hemorrhage
b. Infection
c. Prematurity
F. ABRUPTIO PLACENTA – premature separation of the placenta.
1. Predisposing factors:
a. Maternal hypertension or toxemia
b. Increasing parity and maternal age
c. Sudden release of amniotic fluid
d. Short umbilical cord
e. Hypofibrinoginemia
2. Signs and symptoms:
a. Severe, sharp, knife-like, stabbing pain high in the fundus
b. Hard, beardlike uterus; rigid abdomen
c. Signs of shock
d. Concealed bleeding, if extensive, causes the uterus to lose its ability to contract. It becomes ecchymotic and copper-colored, called Couvelaire uterus, causing severe bleeding. Since the uterus no longer has the ability to contract, hysterectomy will have to be done.
II. TOXEMIA/PREGNANCY-INDUCED HYPERTENSION (PIH) – a vascular disease of unknown cause which occurs anytime after the 24th week of gestation up to 2 weeks post-partum. It has the following triad of symptoms: hypertension, edema and proteinuria (specifically albuminuria).
A. Predisposing factors:
1. Age – primis under 20 and over 30 years
2. Gravida – 5 or more pregnancies
3. Low socioeconomic status (SES)
4. Multiple pregnancy
5. With underlying medical conditions, e.g., heart disease, hypertension or diabetes.
B. Pathogenesis:
C. Diagnosis: roll-over test – assess the probability of developing toxemia when performed between the 28th and 32nd week of pregnancy.
1. Procedure:
a. Patient lies in lateral recumbent position for 15 minutes until BP has stabilized
b. Then rolls over to back position
c. BP is taken at 1 minute and 5 minutes after having rolled over.
2. Interpretation: if diastolic increases 20 mm Hg, or more, patient is prone to toxemia.
Table 10. Classification
I. Acute toxemia – symptoms appear after 24th week of gestation
A. Preeclampsia:
1. Mild
2. Severe
B. Eclampsia
II. Chronic Hypertension with pregnancy
III. Unclassified
D. Details:
1. Preeclampsia
a. Underlying causes:
• Insufficient production of blood and platelets
• Generalized vasoconstriction and associated microangiopathy (-disease of capillaries)
• Abnormal retention of sodium and water by body tissues
b. Medical complications:
• Cerebrovascular hemorrhage
• Acute pulmonary edema
• Acute renal failure
c. Types:
• Mild preeclampsia
• Signs and symptoms:
• Sudden, excessive weight gain of 1-5 lbs per week (earliest sing of preeclampsia) due to edema which is persistent and found in the upper half of the body (e.g., inability to wear the wedding ring)
• Systolic BP of 140, or an increase of 30 mmHg, or more and a diastolic of 90, or a rise of 15 mmHg or more, taken twice, 6 hours apart
• Proteinuria of 0.5 gm/liter or more
• Severe preeclampsia
• Signs and symptoms
• BP of 160/110 mmHg
• Proteinuria of 5 gm/liter or more in 24 hours
• Oliguria of 400 ml or less in 24 hours (normal urine output in 24 hours = 1560 ml)
• Cerebral or visual disturbances
• Pulmonary edema and cyanosis
• Epigastric pain (considered an aura to the development of convulsions)
• Anarsavea/pitting edema; dependent type
• Headache
• Blurred vision
• Oliguria
• Epigastric pain (Aura)
2. Eclampsia – the main difference between preeclampsia and eclampsia is the presence of convulsions in eclampsia.
a. Signs and symptoms – as in preeclampsia plus:
• Increased BUN
• Increased uric acid
• Decreased CO2 combining power
E. Management:
1. Complete bed rest – sodium tends to be excreted at a more rapid rate if the patient is at rest. Energy conservation is important in decreasing metabolic rate to minimize demands for oxygen. Lowered oxygen tension in toxemia is the result of vasoconstriction and decreased blood flow that diminishes the amount of nutrients and oxygen in the cells. In any condition wherein there is a possibility of convulsions, bed rest should be in a darkened, non-stimulating environment with minimal handling.
2. Diet:
a. For mile preeclampsia – high protein, high carbohydrate, moderate salt restriction (no added table salt, (including “bagoong”, “patis” and “toyo”), dired fish (e.g., “daing” and “tuyo”), canned goods, bottled drinks, preserved foods and cold cuts)
b. For severe preeclampsia – highprotein, high carbohydrate and salt-poor (3 gms of salt per day)
3. Medications:
a. Diuretics – hourly urine output should be at least 20-30 ml (normally 50-60 ml per hour). E.g., chlorothiazide/Diuril.
• Pharmacologic effect: decrease reabsorption of sodium and chloride at the proximal tubules, thereby increasing renal excretion of sodium, chloride and water, including potassium.
• Side effects: fatigue and muscle weakness due to fluid and electrolyte imbalance
• Nursing care: closely monitor intake and output
b. Digitalis – if with heart failure
• Pharmacologic action: Increase the force and contraction of the heart, thereby decreasing heart rate. Should not be given, therefore, if heart rate is below 60/minute. (Implication: take the heart rate before giving the drug.)
c. Potassium supplements – any patient receiving diuretics are prone to hypokalemia; if digitalis is given at the same time, hypokalemia increases the sensitivity of the patient to the effects of digitalis. Potassium supplements (e.g. banana) must be given to prevent cardiac arrhythmias.
d. Barbiturates – sedation by means of CNS depression
e. Analgesics: antihypertensives; antibiotics; anticonvulsants
f. Magnesium sulfate – drug of choice
• Actions:
• CNS depressant – lessens possibility of convulsions
• Vasodilator – decreases the BP
• Cathartic – it reduces edema by causing a shift of fluid from the extracellular spaces into the intestines from where the fluid can be excreted
• Dosage: 10 Gms initially, either by slow IV push over 5-10 minutes, or deep IM, 5 Gms/buttock; then IV drip of 1 Gm/hour (1 GM/100 ml D1 xxxxx) IF:
• Deep tendon reflexes are present
• Respiratory rate is at least 12 per minute
• Urine output is at least 100 ml
• Antidote for Magnesium sulfate toxicity: Calcium gluconate 10% IV to maintain cardiac and vascular tone
• Earliest sing of Magnesium sulfate toxicity: disappearance of the knee jerk/patellar reflex
4. Methods of Delivery – preferably vaginal, but it not possible, CS will have to be done
F. Prognosis: the danger of convulsions is present until 48 hours postpartum.
III. DIABETES MELLITUS – chronic hereditary disease which is characterized by hyperglycemia due to a relative insufficiency or lack of insulin from the pancreas which, in turn, leads to abnormalities in the metabolism of carbohydrates, proteins and fats.
A. Diabetogenic effects of pregnancy – many women who had no evidence of diabetes in the past develop abnormalities in glucose tolerance:
1. Decreased renal threshold for sugar that is why it is not unusual to find sugar in the urine of pregnant women
2. Increased production of adrenocorticoids, anterior pituitary hormones and thyroxins which affect carbohydrate and lipoid metabolism, thus increasing carbohydrate concentration in the blood (- hyperglycemia).
3. Rate of insulin secretion is increased BUT sensitivity of the pregnant body to insulin is decreased, i.e., insulin does not seem to be normally effective during pregnancy.
B. Attendant risks:
1. Toxemia
2. Infection
3. Hemorrhage
4. Polyhydramnios
5. Spontaneous abortion – because of vascular complication which affect placental circulation
6. Acidosis – because of nausea and vomiting. Is the chief threat to the fetus in utero.
7. Dystocia – due to excessively large baby
C. Diagnosis – made on the basis of the Glucose Tolerance Test
1. Procedure:
a. NPO after midnight
b. 2 ml of 50% glucose/3 kg of pre-pregnant body weight is given IV (oral tablet not advisable because of known decreased gastric motility and delayed absorption of sugar during pregnancy)
2. Interpretation of results:
a. If less than 100 mg – normal
b. If 100-120 mg% - possible gestational diabetes
c. If more than 120 mg% - overt gestational diabetes
D. Categories – to predict the outcome of pregnancy
1. Class A – GTT is only slightly abnormal; minimal dietary restriction; insulin not needed; fetal survival is high
2. Class C to E – have 25 % prenatal mortality
3. Class F – therapeutic abortion (in other countries) may be justified
E. Management:
1. Diet – highly individualized. Adequate glucose intake (1800-2200 calories) is necessary to prevent intrauterine growth retardation
2. Insulin requirements are likewise highly individualized, requiring close observation throughout pregnancy. Since the effects of the hormones are more pronounced during the second half of pregnancy, the insulin requirements during the 2nd and 3rd trimesters are, therefore, greater.
a. Insulin is regulated to keep urine +1 for sugar (minimal) glycosuria is necessary to prevent acidosis, but negative for acetone
b. Long-acting insulin (Ultralente) will have to be change to regular insulin (Lente) during the last few weeks of pregnancy.
3. Often delivered by CS because:
a. Baby is typically larger or maybe in distress because of placental insufficiency
b. Severe metabolic imbalances in vaginal delivery can occur because of depletion of glycogen reserved in the liver and skeletal muscles by strenuous muscular exertion during labor
4. Maximum difficulty in controlling diabetes is during the early postpartum period because of the drastic changes in hormonal levels.
F. Infant of the Diabetic Mother (IDM)
1. Is typically longer and weighs more because of:
a. Excessive supply of glucose from the mother
b. Increased production of growth hormones from the maternal pituitary
c. Increased secretion of insulin from the fetal pancreas
d. Increased action of adrenocortical hormones that favor passage of glucose from mother to fetus
2. Congenital anomalies are more often seen
3. Cushingoid appearance (puffy, but limp and lethargic)
4. More often born premature, so respiratory distress syndrome is common
5. Lose a greater proportion of weight than normal newborns because of loss of extra fluid
6. Are prone to the following complications:
a. Hypoglycemia – blood sugar level less than 30 mgs. It is the most common complication to watch for
• Cause: while inside the uterus, the fetus tends to be hyperglycemic because of maternal hyperglycemia. The fetal pancreas thus responds to the high glucose level of insulin. Following delivery, the glucose level begins to fall because the baby has been severed from the mother. Since there has been previous production of high levels of insulin, hypoglycemia develops.
• Clinical signs:
• Shrill, high-pitched cry
• Listlessness/jitterness/tremors
• Lethargy; poor suck
• Apnea; cyanosis
• Hypotonia; hypothermia
• Convulsions
• Consequence: hypoglycemia, if not treated, can lead to brain damage and even death
• Management: feed with glucose water earlier than usual or administer IV of glucose
b. Hypocalcemia – serum calcium level of less 7 mg%.
• Signs: same as hypelycemia
• Management: Calcium gluconate to prevent hypocalcemic tetany
IV. HEART DISEASE
A. Classification:
1. Class I – no limitation of physical activity
2. Class II – slight limitation of physical activity; ordinary activity causes fatigue, palpitation, dyspnea or angina
3. Class III – moderate to marked limitation of physical activity; less than ordinary activity causes fatigue, etc.
4. Class IV – unable to carry on any activity without experiencing discomfort
B. Prognosis:
1. Class I and II – normal pregnancy and delivery
2. Class III and IV – poor candidates
C. Signs and symptoms:
1. Because of increased total cardiac volume during pregnancy, heart murmurs are observed.
2. Cardiac output may become so decreased that vital organs are not perfused adequately; oxygen and nutritional requirements are not met.
3. Since the left side of the heart is not able to empty the pulmonary vessels adequately, the latter become engorged, causing pulmonary edema and hypertension. Moist cough in gravidocardiacs, therefore, is a danger sign.
4. Liver and the other organs become congested because blood returning to the heart may not be handled adequately, causing the venous pressure to rise. Fluid then escapes through the walls of engorged capillaries and cause edema or ascites.
5. Congestive heart failure is a high probability also because of the increased cardiac output during pregnancy: dyspnea, exhaustion, edema, pulse irregularities, chest pain on exertion and cyanosis of nailbeds are obvious
D. Management:
1. Bed rest – especially after the 30th week of gestation to ensure that pregnancy is carried to term or at least 36 weeks
2. Diet – should gain enough, but not to much as it would add to the workload of the heart
3. Medications:
a. Digitalis
b. Iron preparations, e.g., Fer-in-sol or Feosol – anemia should be prevented because the body compensates by increasing cardiac output, thus further increasing cardiac workload.
4. Classes III and IV are not put on lithotomy position during delivery to avoid increasing venous return. The semi-sitting position is preferred to facilitate easy respirations.
5. Anesthetic of choice is caudal anesthesia for effortless, pushless and painless delivery. Remember: Gravidocardiacs are not allowed to push with contractions (to prevent Valsalva maneuver which increase venous return to an already weak, damaged heart). Low forceps, therefore, is the best method of delivery.
6. Ergotrate and other oxytocics, scopolamine, diethylstilbestrol and oral contraceptives are contraindicated because they cause fluid retention and promote thromboembolization.
7. Most critical period – the period immediately foll0owing delivery because the 30%-50% increase in blood volume during pregnancy will be reabsorbed into the mother’s circulation in a matter of 5-10 minutes and the weak heart must make rapid adjustment to this change.
VI. MULTIPLE PREGNANCY (Twin Pregnancy)
A. Classification:
1. Monozygotic/Identical – twins begin with a single ovum and sperm, but in the process of fusion or in one of the first cell divisions, the zygote divides into two identical individuals.
a. Characteristics:
• Always of the same sex
• With 2 amnions, 2 chorion, 2 umbilical cords and 2 placentas fused as one
b. Incidence – a chance occurrence:
• More frequent among non-whites
• More frequent among young primis and old multis
2. Dizygotic/Fraternal – two separate ova are fertilized by 2 different sperms. They are actually siblings growing at the same time in utero
a. Characteristics:
• May or may not be of the same sex
• With 2 amnions, 2 chorions, 2 placentas, and 2 umbilical cords
b. Incidence – familial maternal patterns of inheritance
B. Suspect multiple pregnancy if:
1. Faster rate of increase uterine size
2. On quickening, there are several flurries of action in different abdominal positions
3. On auscultation, 2 sets of fetal heart tones are heard
4. There is marked weight gain, not due to toxemia or obesity
C. Compications:
1. Toxemia
2. Polyhydramnios
3. Anemia
4. Abruptic placenta
5. Prematurity
6. Postpartum hemorrhage
VII. INSTRUMENTAL DELIVERIES
A. Forceps Delivery – use of metal instruments (e.g., Simpson, Elliot, Piper for breech presentation) in order to extract the fetus from the birth canal. Forceps are applied when the fetal head is at the perineum (+3 or +4station) and the sagittal suture line is in an anteroposterior position in relation to the outlet.
1. Purposes:
a. Shorten second stage of labor – primary purpose because of:
• Fetal distress
• Maternal exhaustion
• Maternal disease – cardiac, pulmonary complications, hemorrhage
• Ineffective pushing due to anesthesia
b. Prevent excessive pounding of fetal head against the perineum (e.g., low forceps for preemies)
c. Poor uterine contractions or rigid perineum
2. Prerequisites: prolonged and severe stretching
a. Pelvic xxxxxxxxxxxxxxxxxxxxxxxxxxx
b. xxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxx
c. xxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxx
d. Membranes have ruptured
e. Vertical presentation has been established
f. Rectum and bladder are empty
g. Anesthesia is given for sufficient perineal relaxation and to prevent pain
3. Types:
a. Low
b. Mid
4. Complications:
a. Forceps marks –are normal and noticeable only for 24-48 hours
b. Bladder or rectal injury, facial paralysis, ptosis, seizures, epilepsy, cerebral palsy are actually rare
B. Cesarean Section (CS)
1. Indications:
a. Cephalopelvic disproportion (CPD) – most common reason
b. Severe toxemia, placental accidents, fetal distress
c. Previous classic CS – selective CS – done prior to onset of labor pains
2. Types:
a. Low segment – the method of choice. Incision is made in the lower uterine segment which is the thinnest and most passive part during active labor.
• Advantages:
• Minimal blood loss
• Incision is easier to repair
• Lower incidence of postpartum infection
• No possibility of uterine rupture
b. Classic – vertical incision. Recommended in:
• Bladder or lower uterine segment adhesions resulting from previous operations
• Anterior placenta previa
• Transverse lie
3. Preoperative care – patient for CS is both a surgical and an obstetrical patient:
a. Check vital signs, uterine contractions and fetal heart rate
b. Physical examination; routine laboratory tests, blood typing and cross-matching
c. Abdominal is shaved from the level of the xiphoid process/below the nipple line, extending out to the flanks on both sides up to the upper thirds of the thighs.
d. Retention catheter is inserted to constant drainage to keep the bladder away from the operative site.
e. Preoperative medication is usually only Atropine sulfate. No narcotics are given in order to prevent respiratory depression in the newborn.
4. Postoperative care:
a. Deep breathing, coughing exercises; turning from side to side
b. Ambulate after 12 hours
c. Monitor vital signs
d. Watch for signs of hemorrhage – inspect lochia; feel fundus (when boggy, massage with proper abdominal splinting and give analgesics as ordered).
e. Breastfeeding, if desired, should be started 24 hours after delivery (anestheti9c can be transmitted through breastmilk)
5. Most common complication: pelvic thrombosis
VIII. INDUCED LABOR – to bring out labor either by amniotomy or drugs (oxytocin or prostaglandins) before the time when it would have occurred spontaneously or because it does not occur spontaneously.
A. Indications:
1. Maternal
a. Toxemia
b. Placental accidents
c. Premature rupture of the BOW
2. Fetal
a. Diabetes – terminated about 37 weeks GA if indicated
b. Blood incompatibility with rising titer
c. Excessive size
d. Postmaturity
B. Prerequisites:
1. No CPD
2. Fetus is viable – survival is decreased if below 32 weeks CA
3. Single fetus is longitudinal lie and is engaged
4. Ripe cervix – fully or partially effaced; dilated at least 1 – 2 cm
C. Procedure
1. Oxytocin administration:
a. 10 IU of Pitocin in 1000 ml of D5W at a slow rate of 8 gtts/minute given initially. If no fetal distress is observed in 30 minutes, infusion rate is increased 16-20 drops per minute.
b. Amniotomy will be done when cervical dilatation reaches 4 cm. Check FHR and quality of fluid after amniotomy.
c. Nursing care
• Primary concern: monitor intensity of uterine contractions. Remember: if uterine contractions are unduly sustained uterine rupture can occur.
• Monitor flow rate regularly
• Turn off IV drip if with abnormalities in FHR or uterine contractions
• Watch out for:
• Hypertension – oxytocin is a vasoconstrictor
• Antidiuresis leading to water intoxication
• Headache and vomiting
• Convulsions, coma, even death
2. Prostaglandin administration:
a. Route: Either oral or IV, never IM, because it causes tissue irritation
b. Effect: compared to oxytocin, the onset of contraction is slower.
IX. INFECTIONS
A. Syphilis
1. Cause: Treponema pallidum – a spirochete which enters the body during coitus or through cuts and other breaks in the skin or mucous membrane.
2. Treatment: 2.4 – 4.8 million units of Penicillin (if allergic, 30 – 40 gms. of erythrocin) will usually prevent congenital syphilis in the newborn because Penicillin readily crosses the placenta. If untreated, syphilis can cause midtrimester abortion, CNS lesions in the newborn or even death.
3. The newborn with congenital syphilis
a. Signs and symptoms:
• Jaundice at 2 weeks of life – first signs of the disease
• Anemia and hepatosplonomegaly
• “snuffles” (persistent rhinorrhea); coppery rashes on plams and soles; mucous patches; condylomas; pseudoparalysis due to bone inflammation
• If untreated, can progress on to deformed bones, teeth, nose, joints and CNS syphilis
b. Management: Penicillin IM for 10 days or one long-acting Penicillin (Penadur LA)
B. Rubella/German Measles
1. Incidence:
a. Mother – the earlier the mother contacted the disease, the greater the likelihood that the baby will be affected. The rubella virus slows down division of infected cells during organogenesis.
b. Newborn – can carry and transmit the virus for as long as 12-24 months after birth
2. Signs and symptoms of Congenital Rubella Syndrome:
a. Low birth weight; jaundice; petechiae; anemia; thrombocytopenia; hepatosplenomegaly
b. Classes sequelae;
• Eyes: choricretinitis, cataract, glaucoma
• Heart: Patent Ductus arteriosus, stenosis, coarctations
• Xxxx nerve deafness
• Dental and facial clefts
X. BLOOD INCOMPATIBILITY – excessive destruction of fetal red blood cells which occurs when the:
A. Mother is Rh negative and the fetus is Rh positive (because the father is either a homozygous or a heterozygous Rh positive)
B. Mother is Type O and the fetus is either Type A or Type B (because the father is either Type A or B)
XI. MISCELLANEOUS RISK FACTORS
A. Age – maternal and infant mortality rates tend to be high in cases in which the mother is younger than 15 or older than 40.
1. Adolescent pregnancy – is a high-risk pregnancy from both a physical and a psychosocial standpoint. Physical, because of rapid growth of both the pregnant adolescent and her fetus, causing possible depletion of nutritional reserves. Psychosocial, because adolescence is a crisis period by itself, compounded by the situational crisis of pregnancy, plus the fact that most pregnant adolescents are unwed adolescents.
a. Most common problems of pregnant adolescents:
• Toxemia
• Iron-deficiency anemia
2. Advanced age – is a precipitating factor in:
a. Placental accidents
b. Toxemia
c. Uterine atony or inertia
d. Varicosities; hemorrhoids
e. Low birth weight babies
f. Chromosomal abnormalities, e.g., Down’s syndrome/Trisomy 21/Mongolism, commonly associated with menopause:
• Mechanism – a transitional phase, called the elimacterie, heralds the onset of menopause. During this 1-2 year period, the monthly menstrual flow occurs less frequently, is irregular and diminished in amount. Ovulatory and an-ovulatory periods, however, occur (that is why contraceptive methods are advised until the menses have been absent for at least six continuous months). After there have been periods for one year, menopause is said to have occurred.
• Classic signs:
• Vasomotor changes due to hormonal imbalances:
• Hot flushes (head, neck, upper thorax)
• Excessive sweating especially at night
• Emotional changes – insomnia, headache, palpitations, nervousness, apprehension, depression
• Tendency to gain weight more rapidly
• Tendency to lose height because of osteoporosis (“dowager hump”)
• Arthralgias and muscle pains
• Loss of skin elasticity and subcutaneous fat in labial folds
• Artificial menopause/surgically-induced menopause – results from:
• Ocphorectomy or irradiation of ovaries
• Panhysterectomy (more hysterectomy will not lead to menopause since ovaries are still intact; only menstruation will be absent)
B. Parity – first pregnancy is the period of highest risk. Risk increases steadily from gravida 5 and above, especially when the mother is over 40 years of age.
C. Birth interval – a subsequent pregnancy within 3 months of a previous delivery is high risk, as much as a birth interval of more than 5 years.
D. Weight
1. A pre-pregnant weight of less than 70 lbs or more than 180 lbs is a risk factor
2. A weight gain during pregnancy of less than 10 lbs can lead to low birth weight babies, prematurity, abortion, stillbirth and toxemia. A weight gain of more than 30 lbs during pregnancy maybe a sign of toxemia, diabetes, poly-hydramnios, H-mole or multiple pregnancy.
E. Height – a primi of short stature (less than 4 feet, 10 inches) could mean a contracted pelvis or cephalopelvic disproportion.
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