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appendectomy
An appendectomy is the surgical removal of the appendix, a small tube that branches off the large intestine, to treat acute appendicitis. Appendicitis is the acute inflammation of this tube due to infection. -
breast biopsy
A biopsy is a diagnostic test involving the removal of tissue or cells for examination under a microscope. This procedure is also used to remove abnormal breast tissue. A biopsy may be performed using a hollow needle to extract tissue (needle aspiration), or a lump may be partially or completely removed (lumpectomy) for examination and/or treatment. -
carotid endarterectomy
Carotid endarterectomy is a surgical procedure to remove blockage from carotid arteries, the arteries located in the neck that supply blood to the brain. Left untreated, a blocked carotid artery can lead to a stroke. -
cataract surgery
Cataracts cloud the normally clear lens of the eyes. Cataract surgery involves the removal of the cloudy contents with ultrasound waves. In some cases, the entire lens is removed. -
cesarean section
Cesarean section (also called a c-section) is the surgical delivery of a baby by an incision through the mother's abdomen and uterus. This procedure is performed when physicians determine it a safer alternative than a vaginal delivery for the mother, baby, or both. -
cholecystectomy
A cholecystectomy is surgery to remove the gallbladder (a pear-shaped sac near the right lobe of the liver that holds bile). A gallbladder may need to be removed if the organ is prone to troublesome gallstones, if it is infected, or becomes cancerous. -
coronary artery bypass - Most commonly referred to as simply "bypass surgery," this surgery is often performed in people who have angina (chest pain) and coronary artery disease (where plaque has built up in the arteries). During the surgery, a bypass is created by grafting a piece of a vein above and below the blocked area of a coronary artery, enabling blood to flow around the obstruction. Veins are usually taken from the leg, but arteries from the chest may also be used to create a bypass graft.
Click Image to Enlarge -
debridement of wound, burn, or infection
Debridement involves the surgical removal of foreign material and/or dead, damaged, or infected tissue from a wound or burn. By removing the diseased or dead tissue, healthy tissue is exposed to allow for more effective healing. -
dilation and curettage (Also called D & C.)
A D&C is a minor operation in which the cervix is dilated (expanded) so that the cervical canal and uterine lining can be scraped with a curette (spoon-shaped instrument). -
free skin graft
A skin graft involves detaching healthy skin from one part of the body to repair areas of lost or damaged skin in another part of the body. Skin grafts are often performed as a result of burns, injury, or surgical removal of diseased skin. They are most often performed when the area is too large to be repaired by stitching or natural healing. -
hemorrhoidectomy
A hemorrhoidectomy is the surgical removal of hemorrhoids, distended veins in the lower rectum or anus. -
hysterectomy
A hysterectomy is the surgical removal of a woman's uterus. This may be performed either through an abdominal incision or vaginally. -
hysteroscopy
Hysteroscopy is a surgical procedure used to help diagnose and treat many uterine disorders. The hysteroscope (a viewing instrument inserted through the vagina for a visual examination of the canal of the cervix and the interior of the uterus) can transmit an image of the uterine canal and cavity to a television screen. -
inguinal hernia repair
Inguinal hernias are protrusions of part of the intestine into the muscles of the groin. Surgical repair pulls the intestine back to its original location. -
low back pain surgery
Low back pain can have various causes, including abnormal development of the backbone, stress on the back, injury, or a physical disorder that affects the bones of the spine. Usually, surgery is not considered until other options have been exhausted, including rest, medication, and mild exercise. The type of surgery performed on the back depends on the diagnosis. -
mastectomy
A mastectomy is the removal of all or part of the breast. Mastectomies are usually performed to treat breast cancer. There are several types of mastectomies, including the following: -
partial (segmental) mastectomy, involves the removal of the breast cancer and a larger portion of the normal breast tissue around the breast cancer.
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total (or simple) mastectomy, in which the surgeon removes the entire breast, including the nipple, the areola (the colored, circular area around the nipple), and most of the overlying skin, and may also remove some of the lymph nodes under the arm, also called the axillary lymph glands.
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modified radical mastectomy, in which the surgeon removes the entire breast (including the nipple, the areola, and the overlying skin), some of the lymph nodes under the arm, and the lining over the chest muscles. In some cases, part of the chest wall muscles is also removed.
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radical mastectomy, involves removal of the entire breast (including the nipple, the areola, and the overlying skin), the lymph nodes under the arm, and the chest muscles.
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partial colectomy
A partial colectomy is the removal of part of the large intestine (colon) which may be performed to treat cancer of the colon or long-term ulcerative colitis. -
prostatectomy
The surgical removal of all or part of the prostate gland, the sex gland in men that surrounds the neck of the bladder and urethra - the tube that carries urine away from the bladder. A prostatectomy may be performed for an enlarged prostate, benign prostatic hyperplasia (BPH), or if the prostate gland is cancerous. -
releasing of peritoneal adhesions
The peritoneum is a two-layered membrane that lines the wall of the abdominal cavity and covers abdominal organs. Sometimes, organs begin to adhere to the peritoneum, requiring surgery to detach them. -
tonsillectomy
The surgical removal of one or both tonsils. Tonsils are located at the back of the mouth and help fight infections.
Bullets
Common Surgical Procedure
COMMON LAB TESTS FOR HEMATOLOGIC DISORDERS
1. Blood
* Complete blood count (CBC)
* Differential blood count
* Sickle cell trait
* Laboratory tests for Anemias and Leukemias
* Transferrin saturation
* Serum ferritin
* Serum folic acid
* Serum iron
* Serum total iron-binding capacity
* Vitamin B12 assay
* Reticulocyte count
2. Laboratory tests for hemolytic disorders
* Serum haptoglobin
* Hemoglobin electrophoresis
* Osmotic fragility
* RBC enzyme assays
* RBC survival time
3. Laboratory tests for bleeding disorders (illustration )
* Platelet count
* Bleeding time
* Partial thromboplastin time or activated partial thromboplastin time
* Prothrombin time
* Thrombin time
* Prothrombin consumption time
* Platelet aggregation
* Fibrinogen level I
* Fibrinogen degradation
* Antithrombin III
* Coagulation factor assays
* Complete blood count (CBC)
* Differential blood count
* Sickle cell trait
* Laboratory tests for Anemias and Leukemias
* Transferrin saturation
* Serum ferritin
* Serum folic acid
* Serum iron
* Serum total iron-binding capacity
* Vitamin B12 assay
* Reticulocyte count
2. Laboratory tests for hemolytic disorders
* Serum haptoglobin
* Hemoglobin electrophoresis
* Osmotic fragility
* RBC enzyme assays
* RBC survival time
3. Laboratory tests for bleeding disorders (illustration )
* Platelet count
* Bleeding time
* Partial thromboplastin time or activated partial thromboplastin time
* Prothrombin time
* Thrombin time
* Prothrombin consumption time
* Platelet aggregation
* Fibrinogen level I
* Fibrinogen degradation
* Antithrombin III
* Coagulation factor assays
COMMON LAB TESTS FOR THE GENITOURINARY SYSTEM
1. Urine
* culture and sensitivity
* pregnancy
* osmolality
* WBC
* RBC
* casts
* specific gravity
* ketones
* esterase
* nitrates
2. Blood
* serum electrolytes, creatinine, BUN
* osmolality
* CBC
* uric acid
* ESR
* CRPp
* serum mucoprotein
* serum complement
* pregnancy
* estrogen and progesterone levels
* RPR
* HIV assay - ELISA
* antibody responses: ASO, ASKase, AHase, ADNase-B
* prostate-specific antigen
* culture and sensitivity
* pregnancy
* osmolality
* WBC
* RBC
* casts
* specific gravity
* ketones
* esterase
* nitrates
2. Blood
* serum electrolytes, creatinine, BUN
* osmolality
* CBC
* uric acid
* ESR
* CRPp
* serum mucoprotein
* serum complement
* pregnancy
* estrogen and progesterone levels
* RPR
* HIV assay - ELISA
* antibody responses: ASO, ASKase, AHase, ADNase-B
* prostate-specific antigen
COMMON LAB TESTS FOR THE RENAL SYSTEM
1. Blood
* CBC
* Serum chemistry
* Serum electrolytes
* Serum osmolality
* Lipid levels
2. Urine
* pH
* Osmolality
* Specific gravity
* Sodium
* Creatinine
* Sediment
* Creatinine clearance
* Casts
* Culture and sensitivity
* CBC
* Serum chemistry
* Serum electrolytes
* Serum osmolality
* Lipid levels
2. Urine
* pH
* Osmolality
* Specific gravity
* Sodium
* Creatinine
* Sediment
* Creatinine clearance
* Casts
* Culture and sensitivity
COMMON LAB TESTS FOR ENDOCRINE AND HORMONAL DISORDERS
1. Blood or serum
* Supine 10-160 ng/liter
*Upright 8.9-58 ng/dl
* Male: <5 ng/dl
* Female: <10 ng/dl
- 1. Serum electrolytes
- 2. Plasma Fasting ACTH
*8 AM <60 pg/ml
*4 PM 10-50 pg/ml
*4 PM 10-50 pg/ml
- 3.Plasma Aldosterone Concentration (PAC)
* Supine 10-160 ng/liter
*Upright 8.9-58 ng/dl
- 4.Plasma Renin Activity (PRA)
- 5. Plasma Cortisol
- 6.Catecholamines
- 7.Thyroid hormone levels: TSH 0.35-6.20 µU/ml
- 8.Fasting Blood Sugar
- 9.Blood insulin levels
- 10. Growth hormone
* Male: <5 ng/dl
* Female: <10 ng/dl
- 11.Cosyntropin
- 12.ESR
- 13.CRP
- 1. 17-hydroxysteroids
- 2.Free catecholamines
- 3.Osmolality
- 4. Glucose tolerance test
- 3.Karyotyping
- 4.Water deprivation study
COMMON LAB TESTS FOR INTEGUMENTARY DISORDERS
1. Blood
* CBC
* Cultures
* IgE
* Eosinophils
* Titers
* IFA
* ELISA (Enzyme-linked immunosorbent assays)
2. Skin/wound
* Cultures
* Scrapings, smears
* Patch testing
* Microscopic exam
* CBC
* Cultures
* IgE
* Eosinophils
* Titers
* IFA
* ELISA (Enzyme-linked immunosorbent assays)
2. Skin/wound
* Cultures
* Scrapings, smears
* Patch testing
* Microscopic exam
COMMON LAB TESTS FOR MUSCULOSKELETAL DISORDERS:
BLOOD TESTS
1. ESR
2. C-reactive protein
3. CBC
4. Serum cultures
5. Serum Calcium
6. ANA
7. Creatinine
1. ESR
2. C-reactive protein
3. CBC
4. Serum cultures
5. Serum Calcium
6. ANA
7. Creatinine
COMMON LAB TESTS FOR NEUROLOGICAL DISORDERS: CEREBROSPINAL FLUID
1. Appearance colorless, clear
2. Pressure 50-180 mm H2O
3. Protein
4. Cell Count
5. Glucose 50-80 mg/dl
6. Gram stain negative for organisms
7. Culture and sensitivity: no grow
2. Pressure 50-180 mm H2O
3. Protein
- 1. Lumbar 15-50 mg/dl
- 2. Cisternal 15-25 mg/dl
- 3. Ventricular 6-15 mg/dl
4. Cell Count
- 1. RBCs negative
- 2. WBCs 0-5
5. Glucose 50-80 mg/dl
6. Gram stain negative for organisms
7. Culture and sensitivity: no grow
COMMON LAB TESTS FOR RESPIRATORY DISORDERS
1. Blood
1. arterial Blood gases
2. blood cultures
3. hemoglobin
4. hematocrit
5. CBC
6. serum electrolytes
7. RAST
8. immunoglobulins
9. cultures Profile II
2. Urine
1. UA
2. culture and sensitivities
3. casts
3. Throat: sputum culture and sensitivites
4. Skin
1. PPD
COMMON LAB TESTS FOR CARDIOVASCULAR DISORDERS
1. Serum Chemistry
2. Serum Electrolytes
3. Alanine aminotransferase (AST) 5-40 IU/L
4. Creatine kinase CK
* Male 55-170U/L
* Female 30-135 U/L
5. CK - MB (isoenzyme) 0-7 U/L
6. Lactic dehydrogenase (LDH)
* LDH1 22%-36%
* LDH2 35%-46%
* LDH313%-26%
* LDH4 3%-10%
* LDH5 2%-9%
7. CBC
8. Lipid levels
9. Prothrombin time
10. Alkaline phosphatase
11. ESR
12. Arterial Blood Gases
13. Troponin
2. Serum Electrolytes
3. Alanine aminotransferase (AST) 5-40 IU/L
4. Creatine kinase CK
* Male 55-170U/L
* Female 30-135 U/L
5. CK - MB (isoenzyme) 0-7 U/L
6. Lactic dehydrogenase (LDH)
* LDH1 22%-36%
* LDH2 35%-46%
* LDH313%-26%
* LDH4 3%-10%
* LDH5 2%-9%
7. CBC
8. Lipid levels
9. Prothrombin time
10. Alkaline phosphatase
11. ESR
12. Arterial Blood Gases
13. Troponin
COMMON LABORATORY TESTS FOR GASTROINTESTINAL DISORDERS
1. Blood
1. CBC
2. Serum electrolytes
3. Serum chemistry
4. Enzyme-linked immunosorbent assays (ELISA)
5. Serum amylase
6. Differential blood count
7. Prothrombin time
8. ALT, AST, Alanine aminotransferase, LDH
9. Serum bilirubin
10. Glucose
11. Ammonia
12. Serum lipase
13. Alkaline phosphatase
2. Stool
1. Occult blood
2. pH
3. Ova & parasites
4. Qualitative fat
5. Reducing substances
6. Bacterial cultures
7. Vital pathogens
8. Leukocytes
3. Urine
1. Osmolality
2. Sodium
3. Potassium
4. Nitrogen
5. Urobilinogen
6. pH
1. CBC
2. Serum electrolytes
3. Serum chemistry
4. Enzyme-linked immunosorbent assays (ELISA)
5. Serum amylase
6. Differential blood count
7. Prothrombin time
8. ALT, AST, Alanine aminotransferase, LDH
9. Serum bilirubin
10. Glucose
11. Ammonia
12. Serum lipase
13. Alkaline phosphatase
2. Stool
1. Occult blood
2. pH
3. Ova & parasites
4. Qualitative fat
5. Reducing substances
6. Bacterial cultures
7. Vital pathogens
8. Leukocytes
3. Urine
1. Osmolality
2. Sodium
3. Potassium
4. Nitrogen
5. Urobilinogen
6. pH
Common Laboratory Tests according to Systems and Disorders
COMMON LABORATORY TESTS FOR GASTROINTESTINAL DISORDERS
COMMON LAB TESTS FOR CARDIOVASCULAR DISORDERS
COMMON LAB TESTS FOR RESPIRATORY DISORDERS
COMMON LAB TESTS FOR NEUROLOGICAL DISORDERS: CEREBROSPINAL FLUID
COMMON LAB TESTS FOR MUSCULOSKELETAL DISORDERS: BLOOD TESTS
COMMON LAB TESTS FOR INTEGUMENTARY DISORDERS
COMMON LAB TESTS FOR ENDOCRINE AND HORMONAL DISORDERS
COMMON LAB TESTS FOR THE RENAL SYSTEM
COMMON LAB TESTS FOR THE GENITOURINARY SYSTEM
COMMON LAB TESTS FOR HEMATOLOGIC DISORDERS
COMMON LAB TESTS FOR CARDIOVASCULAR DISORDERS
COMMON LAB TESTS FOR RESPIRATORY DISORDERS
COMMON LAB TESTS FOR NEUROLOGICAL DISORDERS: CEREBROSPINAL FLUID
COMMON LAB TESTS FOR MUSCULOSKELETAL DISORDERS: BLOOD TESTS
COMMON LAB TESTS FOR INTEGUMENTARY DISORDERS
COMMON LAB TESTS FOR ENDOCRINE AND HORMONAL DISORDERS
COMMON LAB TESTS FOR THE RENAL SYSTEM
COMMON LAB TESTS FOR THE GENITOURINARY SYSTEM
COMMON LAB TESTS FOR HEMATOLOGIC DISORDERS
MS: GASTROINTESTINAL SYSTEM
I. Anatomy and Physiology
A. Upper gastrointestinal tract
B. Lower gastrointestinal tract
C. Accessory digestive organs
D. Process of digestion
II. Disorders of Stomach and Colon
A. Pernicious anemia
B. Peptic ulcer disease
III. Disorders of the Intestines
A. Inflammatory intestinal diseases
B. Diverticular disease
C. Constipation
D. Diarrhea
E. Bowel obstruction
F. Colon cancer
IV. Disorders of the Liver
A. Hepatitis
B. Hepatitis B
C. Cirrhosis
D. Portal hypertension
E. Ascites
F. Hepatic encephalopathy
V. Disorders Pancreas and Gallbladder
A. Acute pancreatitis
B. Cholecystitis
Points to remember
A. Upper gastrointestinal tract
B. Lower gastrointestinal tract
C. Accessory digestive organs
D. Process of digestion
II. Disorders of Stomach and Colon
A. Pernicious anemia
B. Peptic ulcer disease
III. Disorders of the Intestines
A. Inflammatory intestinal diseases
B. Diverticular disease
C. Constipation
D. Diarrhea
E. Bowel obstruction
F. Colon cancer
IV. Disorders of the Liver
A. Hepatitis
B. Hepatitis B
C. Cirrhosis
D. Portal hypertension
E. Ascites
F. Hepatic encephalopathy
V. Disorders Pancreas and Gallbladder
A. Acute pancreatitis
B. Cholecystitis
Points to remember
MS: RESPIRATORY SYSTEM
I.Quick Review of Anatomy and Physiology of Respiratory System
IIIDisorders of Lower Respiratory System (LRS): Obstructive
IV.LRS Disorders: Restrictive
V.LRS Disorders: Infectious
VI.LRS Disorders: Miscellaneous
>>>>Points to remember
- General Respiratory Anatomy and Physiology
- Upper Respiratory
- Lower Respiratory and Accessory Muscles
- Physiology
IIIDisorders of Lower Respiratory System (LRS): Obstructive
IV.LRS Disorders: Restrictive
V.LRS Disorders: Infectious
VI.LRS Disorders: Miscellaneous
>>>>Points to remember
GIT Bullets
• Most obstructions occur in the small bowel.
• Most large bowel obstructions are caused by cancer.
• Onset of cirrhosis is insidious with symptoms such as anorexia, weight loss, malaise, altered bowel habits, nausea and vomiting.
• Management of cirrhosis is directed towards avoiding complications. This is achieved by maintaining fluid, electrolyte and nutritional balance.
• A client with esophageal varices must be monitored for bleeding (e.g., melena stools, hematemesis, and tachycardia.)
• The rupture of esophageal varices is life threatening and associated with a high mortality rate.
• Pancreatitis is often associated with excessive alcohol ingestion.
• Pancreatic cancer is an insidious disease that often goes undetected until its later stages.
• Diverticula are most common in the sigmoid colon.
• Clients with diverticulosis are often asymptomatic.
• A deficiency in dietary fiber is associated with diverticulitis.
• Colostomies: an ascending colostomy drains liquid feces, is difficult to train and requires daily irrigation; a descending colostomy drains solid feces and can be controlled.
• Frequent liquid stools can be indicative of a fecal impaction or intestinal obstruction.
• Bowel sounds tend to be hyperactive in the early phases of an intestinal obstruction.
• Most large bowel obstructions are caused by cancer.
• Onset of cirrhosis is insidious with symptoms such as anorexia, weight loss, malaise, altered bowel habits, nausea and vomiting.
• Management of cirrhosis is directed towards avoiding complications. This is achieved by maintaining fluid, electrolyte and nutritional balance.
• A client with esophageal varices must be monitored for bleeding (e.g., melena stools, hematemesis, and tachycardia.)
• The rupture of esophageal varices is life threatening and associated with a high mortality rate.
• Pancreatitis is often associated with excessive alcohol ingestion.
• Pancreatic cancer is an insidious disease that often goes undetected until its later stages.
• Diverticula are most common in the sigmoid colon.
• Clients with diverticulosis are often asymptomatic.
• A deficiency in dietary fiber is associated with diverticulitis.
• Colostomies: an ascending colostomy drains liquid feces, is difficult to train and requires daily irrigation; a descending colostomy drains solid feces and can be controlled.
• Frequent liquid stools can be indicative of a fecal impaction or intestinal obstruction.
• Bowel sounds tend to be hyperactive in the early phases of an intestinal obstruction.
Disorders of Pancreas and Gallbladder
- Acute pancreatitis
- Definition/etiology - inflammation of the pancreas
- alcohol ingestion
- gall stones
- drug ingestion
- viral infections
- trauma
- Pathophysiology
- autodigestion from premature activation of pancreatic enzymes
- proteases and lipases, normally active in small intestine, are activated in the pancreas
- phospholipase A digests adipose and parenchymal tissues
- elastase digests elastic fibers of blood vessels, producing bleeding
- amylase digests carbohydrates
- inflammation response occurs from enzyme release
- Findings
- left upper quadrant abdominal pain
- pain worsens after eating and when lying flat
- nausea and vomiting
- fever, agitation, confusion
- hypovolemia and shock
- hemorrhage into retroperitoneal space may produce ecchymosis in flank or around umbilicus
- tachypnea, pulmonary infiltrates, atelectasis from circulating enzymes
- Diagnostics
- elevated enzymes: serum amylase, serum lipase, and urinary amylase
- elevated WBCs, decreased hemoglobin and hematocrit
- elevated LDH and AST (SGOT)
- hyperglycemia
- hypocalcemia
- chest x- ray, CT scan, ultrasound, ERCP
Endoscopy helps diagnose and treat many abdominal (and other) disorders. Here are two endoscopic procedures designed for the abdomen:
Endoscopic retrograde cholangiography (ERCP) outlines the common bile duct and helps diagnose pancreatitis. (If it helps, think of the P in ERCP as pancreatitis and "picture" because ERCP pictures the duct.)
Endoscopic retrograde catheterization of the gallbladder (ERCG) helps diagnose cholecystitis. (Think of the G in ERCG as gallbladder.)
- Complications
- respiratory problems - atelectasis, pneumonia from the immobility imposed by pain
- tetany from decreased calcium levels
- abscess or pseudocyst
- Management
- treat cause
- pain relief - meperidine (Demerol)
- fluid maintenance to prevent shock
- insulin for hyperglycemia
- calcium replacement
- decrease stimulation of pancreas
- NPO-TPN (nothing by mouth; total parenteral nutrition)
- NG tube
- anticholinergics
- h2-receptor antagonists
- Nursing interventions
- manage pain
- monitor alteration in breathing patterns
- monitor nutritional status
- oral care when NPO
- if eating is allowed, diet high in proteins and carbohydrates and low in fat
- monitor fluid and electrolyte balances
PANCREATITIS
- Acute pancreatitis can become chronic
- Morphine sulfate is not used to treat pain since it can cause the sphincter of Oddi to spasm
- Pain may be relieved by side-lying
- If clients lose pancreatic function, they may have to take pancreatic enzymes and bile salts with meals.
- With pancreatitis, avoid stimulation of the pancreas: do not use enteral feedings
- Cholecystitis
- Definition/etiology - inflammation of the gallbladder
- usually due to gallstones (Cholelithiasis)
- types
- cholesterol - most common
- pigment - unconjugated bilirubin
- bile is blocked, and infects tissue
- more common in women, especially those over 40 and those who use birth control pills
- Pathophysiology
- common bile duct is obstructed by a gallstone
- bile cannot be excreted, some is reabsorbed
- remaining bile distends and inflames gall bladder
- may scar gallbladder, resulting in less storing of the bile from the liver
- can perforate gall bladder
- Findings
- colicky pain in right upper quadrant with possible radiation to right shoulder and back
- indigestion after eating fatty foods
- nausea and vomiting
- jaundice (if the liver is involved or inflamed or the common duct obstructed)
- low grade fever
- Diagnostics
- endoscopic retrograde cholangiography (ERCP)
- endoscopic retrograde catheterization of the gallbladder (ERCG)
- ultrasound
- Management
- rest
- low-fat diet
- removal of stone in common duct by endoscopy
- to dissolve cholesterol stones
- chenodeoxycholic acid (Chenodiol) - side effects are diarrhea and hepatotoxicity
- ursodeoxycholic acid (UDCA)
- control pain - meperidine (Demerol) is drug of choice
- replace vitamin K if bleeding time is prolonged
- extracorporeal shock wave lithotripsy - may have hematuria after procedure, but not longer than 24 hours
- choledocholithotomy - to remove or break up stones
- laparoscopic laser cholecystectomy
- cholecystectomy
- Nursing interventions
- monitor vital signs
- monitor pain and medicate as needed
- teach client - dietary restriction of fatty foods
STRESS RESPONSE
A. Local responses to stress: Local Adaptation Syndrome (LAS):
1. Examples
a. Blood clotting
b. Wound healing
c. Reflex pain response
d. Inflammatory response
2. Characteristics
a. Localized response
b. Adaptive (that is, requires a stressor)
c. Short-term
d. Restorative
B. Whole-body response to stress: General Adaptation Syndrome (GAS)
1. Involves primarily nervous and endocrine systems, in 3 stages
2. Stage 1: Alarm reaction - exposure to adverse stimulus; body mobilized to resist in form of compensatory behavior
a. Fight or flight response
1. Increased cardiac output
2. Increased heart rate
3. Increased respiratory rate
4. Pupils dilate
5. Increased mental alertness
b. Sympathetic Nervous System response
1. Increased epinephrine
a. Increased heart rate
b. Increased oxygen intake
c. Increased blood sugar
2. Increased norepinephrine
a. Increased blood flow to skeletal muscle
b. Involves increased arterial blood pressure
c. Posterior Pituitary: Increased ADH
1. Increased water reabsorption
2. Decreased urine output
d. Anterior Pituitary: Increased ACTH
1. Increased cortisol secretion
a. Body turns fat and proteins into glycogen
b. Increased protein catabolism
c. Increased fat catabolism
2. Increased aldosterone secretion:
a. Body reabsorbs more sodium, more water
b. Kidneys produce less urine
c. Kidneys secrete more potassium
3. Stage 2: Resistance - When stimulus is excessive or prolonged, alarm and mobilization give way to resistance
a. Stabilization
b. Hormonal levels return to normal
c. Parasympathetic nervous system activates
d. Body adapts to stressors
4. Stage 3: Exhaustion - If stressor continues, energy wanes and body weakens
a. Physiological response as noted in alarm reaction
b. Decreased energy levels
c. Decreased physiologic adaptation
d. Death
1. Examples
a. Blood clotting
b. Wound healing
c. Reflex pain response
d. Inflammatory response
2. Characteristics
a. Localized response
b. Adaptive (that is, requires a stressor)
c. Short-term
d. Restorative
B. Whole-body response to stress: General Adaptation Syndrome (GAS)
1. Involves primarily nervous and endocrine systems, in 3 stages
2. Stage 1: Alarm reaction - exposure to adverse stimulus; body mobilized to resist in form of compensatory behavior
a. Fight or flight response
1. Increased cardiac output
2. Increased heart rate
3. Increased respiratory rate
4. Pupils dilate
5. Increased mental alertness
b. Sympathetic Nervous System response
1. Increased epinephrine
a. Increased heart rate
b. Increased oxygen intake
c. Increased blood sugar
2. Increased norepinephrine
a. Increased blood flow to skeletal muscle
b. Involves increased arterial blood pressure
c. Posterior Pituitary: Increased ADH
1. Increased water reabsorption
2. Decreased urine output
d. Anterior Pituitary: Increased ACTH
1. Increased cortisol secretion
a. Body turns fat and proteins into glycogen
b. Increased protein catabolism
c. Increased fat catabolism
2. Increased aldosterone secretion:
a. Body reabsorbs more sodium, more water
b. Kidneys produce less urine
c. Kidneys secrete more potassium
3. Stage 2: Resistance - When stimulus is excessive or prolonged, alarm and mobilization give way to resistance
a. Stabilization
b. Hormonal levels return to normal
c. Parasympathetic nervous system activates
d. Body adapts to stressors
4. Stage 3: Exhaustion - If stressor continues, energy wanes and body weakens
a. Physiological response as noted in alarm reaction
b. Decreased energy levels
c. Decreased physiologic adaptation
d. Death
Stress Management
A. Stress: a universal phenomenon, stress requires change or adaptation so that the person can maintain equilibrium
B. Stress can be internal or external
C. Nature of stressor involves:
F. Stress response involves both localized and general adaptation
G. Factors affecting stress response
1. Personal: heredity, gender, race, age personality, cognitive ability
2. Sociocultural: finances, support systems
3. Interpersonal: self-esteem, prior coping mechanisms
4. Spiritual: belief system
5. Environmental: crowding, pollution, climate
6. Occupational: work overload, conflict, risk
H. Physiologic indicators of stress
I. Emotional/behavioral indicators of stress
J. Stress can cause a variety of emotional and physical disorders
K. Stress management strategies
L. The non-compliant client does not cooperate with the treatment plan
1. Behavior characteristics
a. does not take prescrived medication
b. continues activities restricted by provider of care, such as smoking
c. does not follow prescribed activities, such as exercise
2. Nursing interventions
a. explore the reasons for non-compliance
i. lack of understanding - reinforce teaching
ii. lack of family support - involve family and support groups
iii. side effects - refer to provider of care
iv. finances and access - refer to Social Services
v. negative attitude toward treatment - encourage expression
b. express genuine concern for client
c. discuss imporvement potential
B. Stress can be internal or external
C. Nature of stressor involves:
- 1. Intensity
- 2. Scope
- 3. Duration
- 4. Other stressors: their number and nature
- 1. Physical - drugs or alcohol
- 2. Psychological - such as adolescent emotional upheaval, or unexpressed anger
- 3. Social - isolation, interpersonal loss
- 4. Cultural - ideal body image
- 5. Microbiologic - infection
F. Stress response involves both localized and general adaptation
G. Factors affecting stress response
1. Personal: heredity, gender, race, age personality, cognitive ability
2. Sociocultural: finances, support systems
3. Interpersonal: self-esteem, prior coping mechanisms
4. Spiritual: belief system
5. Environmental: crowding, pollution, climate
6. Occupational: work overload, conflict, risk
H. Physiologic indicators of stress
- Increased Blood Pressure
- Tachycardia
- Tachypnea
- Sweaty palms
- Cold Hands and Feet
- Decreased urine output
- Dilated Pupils
- Change in appetite
- Gastrointestinal changes: nausea, vomiting, diarrhea
- Headache
- Restlessness
- Insomnia
- Muscle tension
I. Emotional/behavioral indicators of stress
- Behavior Patterns
- Substance use/abuse
- Changes in eating habits
- Changes in activity
- Mood
- Loss of self esteem
- Feelings of inadequacy
- Increased irritability
- Crying
- Cognitive
- Lack of motivation
- Forgetfulness
- Tendency to make mistakes
- Decreased productivity
- Poor judgment
- Inability to concentrate
- Preoccupation
J. Stress can cause a variety of emotional and physical disorders
- Hypertension
- Ulcers
- Skin Disorders
- Cardiovascular disorders
- Increased cholesterol
- Migraines
- Eating Disorders
- Depression
- Substance Abuse
- Asthma
- Cancer
- Rheumatoid Arthritis
- Anxiety disorders
- Dysrhythmias
- Muscle tension/aches
- Sleeping disorders
- Gastrointestinal upset/disorders
- Endocrine disorders
K. Stress management strategies
L. The non-compliant client does not cooperate with the treatment plan
1. Behavior characteristics
a. does not take prescrived medication
b. continues activities restricted by provider of care, such as smoking
c. does not follow prescribed activities, such as exercise
2. Nursing interventions
a. explore the reasons for non-compliance
i. lack of understanding - reinforce teaching
ii. lack of family support - involve family and support groups
iii. side effects - refer to provider of care
iv. finances and access - refer to Social Services
v. negative attitude toward treatment - encourage expression
b. express genuine concern for client
c. discuss imporvement potential
FOUR THEORETIC MODELS OF GRIEF
A. Elizabeth Kubler-Ross: Five Stages DABDA
1. Denial
a. Unconscious avoidance which varies from a brief period to the remainder of life
b. Allows one to mobilize defenses to cope
c. Positive adaptive responses - verbal denial; crying
d. Maladaptive responses - no crying, no acknowledgement of loss
2. Anger
a. Expresses the realization of loss
b. May be overt or covert
c. Positive adaptive responses - verbal expressions of anger
d. Maladaptive responses - persistent guilt or low self esteem, aggression, self destructive ideation or behavior
3. Bargaining
a. An attempt to change reality of loss; person bargains for treatment control, expresses wish to be alive for specific events in near future
b. Maladaptive responses - bargains for unrealistic activities or events in distant future
4. Depression and Withdrawal
a. Sadness resulting from actual and/or anticipated loss
b. Positive adaptive response - crying, social withdrawal
c. Maladaptive responses - self-destructive actions, despair
5. Acceptance
a. Resolution of feelings about death or other loss, resulting in peaceful feelings
b. Positive adaptive behaviors - may wish to be alone, limit social contacts, complete personal business
B. John Bowlby: Four Stages of Separation and Loss
1. Shock
2. Despair
3. Detachment
4. Resolution
C. E. Lindemann
1. Shock
2. Acute mourning
3. Resolution of grief
D. J.W. Wooden
1. Accepting the reality
2. Experiencing the pain
3. Adjusting to the changed environment
4. Withdrawing and reinvesting emotional energy
1. Denial
a. Unconscious avoidance which varies from a brief period to the remainder of life
b. Allows one to mobilize defenses to cope
c. Positive adaptive responses - verbal denial; crying
d. Maladaptive responses - no crying, no acknowledgement of loss
2. Anger
a. Expresses the realization of loss
b. May be overt or covert
c. Positive adaptive responses - verbal expressions of anger
d. Maladaptive responses - persistent guilt or low self esteem, aggression, self destructive ideation or behavior
3. Bargaining
a. An attempt to change reality of loss; person bargains for treatment control, expresses wish to be alive for specific events in near future
b. Maladaptive responses - bargains for unrealistic activities or events in distant future
4. Depression and Withdrawal
a. Sadness resulting from actual and/or anticipated loss
b. Positive adaptive response - crying, social withdrawal
c. Maladaptive responses - self-destructive actions, despair
5. Acceptance
a. Resolution of feelings about death or other loss, resulting in peaceful feelings
b. Positive adaptive behaviors - may wish to be alone, limit social contacts, complete personal business
B. John Bowlby: Four Stages of Separation and Loss
1. Shock
2. Despair
3. Detachment
4. Resolution
C. E. Lindemann
1. Shock
2. Acute mourning
3. Resolution of grief
D. J.W. Wooden
1. Accepting the reality
2. Experiencing the pain
3. Adjusting to the changed environment
4. Withdrawing and reinvesting emotional energy
Grief
A. Loss
1. A universal phenomenon; it occurs across the lifespan
2. There are many types of loss
a. loss of external objects
b. loss of significant other: through death, divorce
c. loss of environment: by moving, taking a new job, hospitalization
d. loss of an aspect of self: may include a body part, physiologic or psychologic function
3. Response to loss depends on
a. one's personality
b. culture
c. previous experience with loss
d. one's values
e. perceived value of loss
f. support system
B. Types of Grief
1. Anticipatory grief: person learns of impending loss and responds with processes of mourning, coping, interaction, planning, and psychosocial reorganization
2. Disenfranchised grief: person experiences a loss that is not or cannot be openly acknowledged, publicly mourned, or socially supported
3. Mourning: process used to resolve grief
4. Tasks of mourning (common to the models of grief) spell R-E-A-L
a. Real: accept that the loss is real
b. Experience the emotions associated with the loss
c. Adjust or re-adjust to life and activities
d. Let go: move on with one's own life
5. Grief theory models
C. Nursing care in grief
1. Support client's effective coping mechanisms
2. Don't take client's responses personally
3. Listen attentively
4. Help client with problem solving and decision making as indicated
5. Encourage the client and/or significant others to ventilate
6. Utilize therapeutic touch as appropriate
7. Assist in discussions of future plans as appropriate
1. A universal phenomenon; it occurs across the lifespan
2. There are many types of loss
a. loss of external objects
b. loss of significant other: through death, divorce
c. loss of environment: by moving, taking a new job, hospitalization
d. loss of an aspect of self: may include a body part, physiologic or psychologic function
3. Response to loss depends on
a. one's personality
b. culture
c. previous experience with loss
d. one's values
e. perceived value of loss
f. support system
B. Types of Grief
1. Anticipatory grief: person learns of impending loss and responds with processes of mourning, coping, interaction, planning, and psychosocial reorganization
2. Disenfranchised grief: person experiences a loss that is not or cannot be openly acknowledged, publicly mourned, or socially supported
3. Mourning: process used to resolve grief
4. Tasks of mourning (common to the models of grief) spell R-E-A-L
a. Real: accept that the loss is real
b. Experience the emotions associated with the loss
c. Adjust or re-adjust to life and activities
d. Let go: move on with one's own life
5. Grief theory models
C. Nursing care in grief
1. Support client's effective coping mechanisms
2. Don't take client's responses personally
3. Listen attentively
4. Help client with problem solving and decision making as indicated
5. Encourage the client and/or significant others to ventilate
6. Utilize therapeutic touch as appropriate
7. Assist in discussions of future plans as appropriate
Therapeutic Communication
- Characterizes the Nurse-Client Relationship
A. Nurse-client relationship: a therapeutic professional relationship in which two people interact
1. The nurse who possesses the skills and ability to provide counseling, crisis intervention, health teaching, etc. and
2. The client who seeks help for some problem
B. Phases of the nurse-client relationship
A. Initiating or orientation
1. Sets time, place and duration of sessions
2. Establishes boundaries of the relationship
3. Identifies the problem and expectations-that is, goal setting
4. Usually an anxious time for both client and nurse
a. Client may be late for the session
b. Client may exhibit anxious mannerisms
c. Nurse's own anxiety may prompt nurse to use techniques that block communication
B. Working
1. Boundaries of the relationship are accepted by the client and the nurse and a therapeutic relationship is established
2. Nurse uses interpersonal skills to communicate with the client
3. Client identifies problems, develops insights to the problems
4. Client learns adaptive coping skills and problem solving
C. Termination
1. Actually begins with the first session and ends when identified treatment goals are met
2. Anticipate problems of termination
a. Client may become too dependent on nurse
b. Client may recall previous separation experiences, and feelings of rejection, depression, and/or abandonment
3. Client and nurse summarize and evaluate work
4. Client and nurse express thoughts and feelings about termination
C. Five characteristics of nurse-client relationship
1. Mutual definition: together, nurse and client define relationship
2. Goal direction: purpose, time, and place are specific
3. Specified boundaries: in time, space, content, and confidentiality
4. Therapeutic communication: nurse eases trust and open communication by these interpersonal techniques
5. Nurse helps client toward resolution
(many Good Scholars Take Nursing)
D. Therapeutic communication
1. Consider the developmental level, culture, and physical condition of the client
2. Focus not on subjective inferences but on actual objective behaviors
3. Focus not on judgment but on description
4. Instead of offering advice and solutions, share information and explore alternatives
5. Focus not on "why" but on how and what
6. For confused or disoriented clients, focus on reality orientation
7. Ask open-ended questions and seek information
8. Focus on nursing interventions
9. To ease this process, use specific techniques
10. Certain techniques block therapeutic communication
THERAPEUTIC COMMUNICATION TECHNIQUES
Not Necessarily Verbal
1. Acceptance - Recognizing the other person without inserting your own values or judgments. May be verbal or nonverbal; with or without understanding
2. Listening - Consciously receiving the client's message. Includes listening actively, responsibly, and seriously
3. Empathy - Experiencing another's feeling temporarily; truly being with and understanding another through active listening
4. Silence - Suspending talk for a therapeutic reason
5. Neutral response - Showing interest and involvement without saying anything else
6. Eye contact - As appropriate to the client's culture
Verbal
7. Self-disclosure - Sharing personal information at an opportune moment to convey understanding or to role model behavior
8. Clarification - Putting into words vague ideas or unclear thoughts of the client. Purpose is to help nurse understand, or invite the client to explain
9. Restating - Repeating to the client the main thought he has expressed to indicate the nurse is listening and interested. May encourage the client to elaborate
10. Refocusing - Picking up on central topics or "cues" given by the client
11. Open-ended questions - Asking questions that cannot be answered "yes" or "no." Used to broaden conversational opportunities and to enable the client to communicate.
12. Incomplete sentences - Encouraging the client to continue with phrases such as "Go on…"
13. Focusing - Helping the client to explore a specific topic
NINE INEFFECTIVE COMMUNICATION TECHNIQUES
1. Giving advice - Telling the client what to do. Giving an opinion or making decisions for the client. Implies the client cannot handle life decisions and that the nurse is accepting responsibility for client.
2. False reassurance - Using clichés, pat answers, cheery words and comforting statements as an attempt to reassure client.
3. Changing the Subject - Introducing new topics inappropriately. May result from poor listening skills
4. Social Response - Responding in a way that either focuses attention on the nurse instead of the client, or is not goal-directed on behalf of the client.
5. Invalidation - Ignoring or denying the client's thoughts or feelings.
6. Overloading - Talking rapidly, changing subjects or asking for more information than can be absorbed at one time; for example, asking two questions at once.
7. Underloading - Remaining silent and unresponsive, not picking up cues and failing to give feedback.
8. Incongruence - Sending verbal and nonverbal messages that contradict one another; often called a double message.
9. Value Judgments - Giving one's own opinion, evaluating , moralizing or implying one's own values by using words such as "should," "ought," "good," or "bad."
E. Other types of therapeutic interventions
1. Group therapy
2. Family therapy
3. Milieu therapy - A method of psychotherapy that controls the environment of the patient to provide interpersonal contacts that will develop trust, assurance, and personal autonomy
A. Nurse-client relationship: a therapeutic professional relationship in which two people interact
1. The nurse who possesses the skills and ability to provide counseling, crisis intervention, health teaching, etc. and
2. The client who seeks help for some problem
B. Phases of the nurse-client relationship
A. Initiating or orientation
1. Sets time, place and duration of sessions
2. Establishes boundaries of the relationship
3. Identifies the problem and expectations-that is, goal setting
4. Usually an anxious time for both client and nurse
a. Client may be late for the session
b. Client may exhibit anxious mannerisms
c. Nurse's own anxiety may prompt nurse to use techniques that block communication
B. Working
1. Boundaries of the relationship are accepted by the client and the nurse and a therapeutic relationship is established
2. Nurse uses interpersonal skills to communicate with the client
3. Client identifies problems, develops insights to the problems
4. Client learns adaptive coping skills and problem solving
C. Termination
1. Actually begins with the first session and ends when identified treatment goals are met
2. Anticipate problems of termination
a. Client may become too dependent on nurse
b. Client may recall previous separation experiences, and feelings of rejection, depression, and/or abandonment
3. Client and nurse summarize and evaluate work
4. Client and nurse express thoughts and feelings about termination
C. Five characteristics of nurse-client relationship
1. Mutual definition: together, nurse and client define relationship
2. Goal direction: purpose, time, and place are specific
3. Specified boundaries: in time, space, content, and confidentiality
4. Therapeutic communication: nurse eases trust and open communication by these interpersonal techniques
5. Nurse helps client toward resolution
(many Good Scholars Take Nursing)
D. Therapeutic communication
1. Consider the developmental level, culture, and physical condition of the client
2. Focus not on subjective inferences but on actual objective behaviors
3. Focus not on judgment but on description
4. Instead of offering advice and solutions, share information and explore alternatives
5. Focus not on "why" but on how and what
6. For confused or disoriented clients, focus on reality orientation
7. Ask open-ended questions and seek information
8. Focus on nursing interventions
9. To ease this process, use specific techniques
10. Certain techniques block therapeutic communication
THERAPEUTIC COMMUNICATION TECHNIQUES
Not Necessarily Verbal
1. Acceptance - Recognizing the other person without inserting your own values or judgments. May be verbal or nonverbal; with or without understanding
2. Listening - Consciously receiving the client's message. Includes listening actively, responsibly, and seriously
3. Empathy - Experiencing another's feeling temporarily; truly being with and understanding another through active listening
4. Silence - Suspending talk for a therapeutic reason
5. Neutral response - Showing interest and involvement without saying anything else
6. Eye contact - As appropriate to the client's culture
Verbal
7. Self-disclosure - Sharing personal information at an opportune moment to convey understanding or to role model behavior
8. Clarification - Putting into words vague ideas or unclear thoughts of the client. Purpose is to help nurse understand, or invite the client to explain
9. Restating - Repeating to the client the main thought he has expressed to indicate the nurse is listening and interested. May encourage the client to elaborate
10. Refocusing - Picking up on central topics or "cues" given by the client
11. Open-ended questions - Asking questions that cannot be answered "yes" or "no." Used to broaden conversational opportunities and to enable the client to communicate.
12. Incomplete sentences - Encouraging the client to continue with phrases such as "Go on…"
13. Focusing - Helping the client to explore a specific topic
NINE INEFFECTIVE COMMUNICATION TECHNIQUES
1. Giving advice - Telling the client what to do. Giving an opinion or making decisions for the client. Implies the client cannot handle life decisions and that the nurse is accepting responsibility for client.
2. False reassurance - Using clichés, pat answers, cheery words and comforting statements as an attempt to reassure client.
3. Changing the Subject - Introducing new topics inappropriately. May result from poor listening skills
4. Social Response - Responding in a way that either focuses attention on the nurse instead of the client, or is not goal-directed on behalf of the client.
5. Invalidation - Ignoring or denying the client's thoughts or feelings.
6. Overloading - Talking rapidly, changing subjects or asking for more information than can be absorbed at one time; for example, asking two questions at once.
7. Underloading - Remaining silent and unresponsive, not picking up cues and failing to give feedback.
8. Incongruence - Sending verbal and nonverbal messages that contradict one another; often called a double message.
9. Value Judgments - Giving one's own opinion, evaluating , moralizing or implying one's own values by using words such as "should," "ought," "good," or "bad."
E. Other types of therapeutic interventions
1. Group therapy
2. Family therapy
3. Milieu therapy - A method of psychotherapy that controls the environment of the patient to provide interpersonal contacts that will develop trust, assurance, and personal autonomy
Coping Mechanisms
(also called defense mechanisms)
A. Definition: psychological techniques that the personality develops to manage anxiety, aggression, hostility, etc.
B. Coping mechanisms represent conflicts between the id and superego
C. Used by both mentally healthy and ill individuals
D. May be used consciously, but are usually unconscious
E. Types of coping mechanisms
1. Compensation - extra effort in one area to offset real or imagined lack in another area
o Example: Short man becomes assertively verbal and excels in business.
2. Conversion - A mental conflict is expressed through physical symptoms
o Example: Woman becomes blind after seeing her husband with another woman.
3. Denial - treating obvious reality factors as though they do not exist because they are consciously intolerable
o Example: Mother refuses to believe her child has been diagnosed with leukemia. "She just has the flu."
4. Displacement - transferring unacceptable feelings aroused by one object to another, more acceptable substitute
o Example: Adolescent lashes out at parents after not being invited to party.
5. Dissociation - walling off specific areas of the personality from consciousness
o Example: Adolescent talks about failing grades as if they belong to someone else; jokes about them.
6. Fantasy - a conscious distortion of unconscious wishes and need to obtain satisfaction
o Example: A student nurse fails the critical care exam and daydreams about her heroic role in a cardiac arrest.
7. Fixation - becoming stagnated in a level of emotional development in which one is comfortable
o Example: A sixty year old man who dresses and acts as if he were still in the 1960's.
8. Identification - subconsciously attributing to oneself qualities of others
o Example: Elvis impersonators.
9. Intellectualization - use of thinking, ideas, or intellect to avoid emotions
o Example: Parent becomes extremely knowledgeable about child's diabetes.
10. Introjection - incorporating the traits of others
o Example: Husband's symptoms mimic wife's before she died.
11. Projection - unconsciously projecting one's own unacceptable qualities or feelings onto others
o Example: Woman who is jealous of another woman's wealth accuses her of being a gold-digger.
12. Rationalization - justifying behaviors, emotions, motives, considered intolerable through acceptable excuses
o Example: "I didn't get chosen for the team because the coach plays favorites."
13. Reaction Formation - expressing unacceptable wishes or behavior by opposite overt behavior
o Example: Recovered smoker preaches about the dangers of second hand smoke.
14. Regression - retreating to an earlier and more comfortable emotional level of development
o Example: Four year old insists on climbing into crib with younger sibling.
15. Repression - unconscious, deliberate forgetting of unacceptable or painful thoughts, impulses, feelings or acts
o Example: Adolescent "forgets" appointment with counselor to discuss final grades.
16. Sublimation - diversion of unacceptable instinctual drives into personally and socially acceptable areas.
o Example: Young woman who hated school becomes a teacher.
A. Definition: psychological techniques that the personality develops to manage anxiety, aggression, hostility, etc.
B. Coping mechanisms represent conflicts between the id and superego
C. Used by both mentally healthy and ill individuals
D. May be used consciously, but are usually unconscious
E. Types of coping mechanisms
1. Compensation - extra effort in one area to offset real or imagined lack in another area
o Example: Short man becomes assertively verbal and excels in business.
2. Conversion - A mental conflict is expressed through physical symptoms
o Example: Woman becomes blind after seeing her husband with another woman.
3. Denial - treating obvious reality factors as though they do not exist because they are consciously intolerable
o Example: Mother refuses to believe her child has been diagnosed with leukemia. "She just has the flu."
4. Displacement - transferring unacceptable feelings aroused by one object to another, more acceptable substitute
o Example: Adolescent lashes out at parents after not being invited to party.
5. Dissociation - walling off specific areas of the personality from consciousness
o Example: Adolescent talks about failing grades as if they belong to someone else; jokes about them.
6. Fantasy - a conscious distortion of unconscious wishes and need to obtain satisfaction
o Example: A student nurse fails the critical care exam and daydreams about her heroic role in a cardiac arrest.
7. Fixation - becoming stagnated in a level of emotional development in which one is comfortable
o Example: A sixty year old man who dresses and acts as if he were still in the 1960's.
8. Identification - subconsciously attributing to oneself qualities of others
o Example: Elvis impersonators.
9. Intellectualization - use of thinking, ideas, or intellect to avoid emotions
o Example: Parent becomes extremely knowledgeable about child's diabetes.
10. Introjection - incorporating the traits of others
o Example: Husband's symptoms mimic wife's before she died.
11. Projection - unconsciously projecting one's own unacceptable qualities or feelings onto others
o Example: Woman who is jealous of another woman's wealth accuses her of being a gold-digger.
12. Rationalization - justifying behaviors, emotions, motives, considered intolerable through acceptable excuses
o Example: "I didn't get chosen for the team because the coach plays favorites."
13. Reaction Formation - expressing unacceptable wishes or behavior by opposite overt behavior
o Example: Recovered smoker preaches about the dangers of second hand smoke.
14. Regression - retreating to an earlier and more comfortable emotional level of development
o Example: Four year old insists on climbing into crib with younger sibling.
15. Repression - unconscious, deliberate forgetting of unacceptable or painful thoughts, impulses, feelings or acts
o Example: Adolescent "forgets" appointment with counselor to discuss final grades.
16. Sublimation - diversion of unacceptable instinctual drives into personally and socially acceptable areas.
o Example: Young woman who hated school becomes a teacher.
Psychiatric Nursing
I. Coping Mechanisms
II. Therapeutic Communication
III. Grief
IV. Stress Management
V. Schizophrenia
VI. Mood Disorders
VII. Anxiety Disorders
VIII. Borderline Personality Disorder
IX. Suicide Prevention
X. Crisis Intervention
XI. Substance Abuse
XII. Autism
XIII. Abuse Syndromes
XIV. Eating Disorders
POINTS to Remember
II. Therapeutic Communication
III. Grief
IV. Stress Management
V. Schizophrenia
VI. Mood Disorders
VII. Anxiety Disorders
VIII. Borderline Personality Disorder
IX. Suicide Prevention
X. Crisis Intervention
XI. Substance Abuse
XII. Autism
XIII. Abuse Syndromes
XIV. Eating Disorders
POINTS to Remember
CRANIOCEREBRAL TRAUMA (ACUTE REHABILITATIVE PHASE)
Craniocerebral trauma, also called head or brain injury (open or closed), includes skull fractures, brain concussion, cerebral contusion/laceration, and hemorrhage (subarachnoid, subdural, epidural, intracerebral, brainstem). Primary injury occurs from a direct or indirect blow to the head, causing acceleration/deceleration of the brain. Secondary brain injury results from diffuse intracerebral axonal injury, intracranial hypertension, hypoxemia, hypercapnia, or systemic hypotension. Cerebral concussion is the most common form of head injury.
Consequences of brain injury range from no apparent neurological disturbance to a persistent vegetative state or death. Therefore, every head injury must be considered potentially dangerous.
CARE SETTING
This plan of care focuses on acute care and acute inpatient rehabilitation. Brain injury care for those experiencing moderate to severe trauma progresses along a continuum of care, beginning with acute inpatient hospital care and inpatient rehabilitation to subacute and outpatient rehabilitation, as well as home- and community-based services.
RELATED CONCERNS
Cerebrovascular accident (CVA)/stroke
Psychosocial aspects of care
Seizure disorders/epilepsy
Surgical intervention
Thrombophlebitis: deep vein thrombosis
Total nutritional support: parenteral/enteral feeding
Upper gastrointestinal/esophageal bleeding
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Consequences of brain injury range from no apparent neurological disturbance to a persistent vegetative state or death. Therefore, every head injury must be considered potentially dangerous.
CARE SETTING
This plan of care focuses on acute care and acute inpatient rehabilitation. Brain injury care for those experiencing moderate to severe trauma progresses along a continuum of care, beginning with acute inpatient hospital care and inpatient rehabilitation to subacute and outpatient rehabilitation, as well as home- and community-based services.
RELATED CONCERNS
Cerebrovascular accident (CVA)/stroke
Psychosocial aspects of care
Seizure disorders/epilepsy
Surgical intervention
Thrombophlebitis: deep vein thrombosis
Total nutritional support: parenteral/enteral feeding
Upper gastrointestinal/esophageal bleeding
Download whole document
CIRRHOSIS OF THE LIVER
Cirrhosis is a chronic disease of the liver characterized by alteration in structure, degenerative changes and widespread destruction of hepatic cells, impairing cellular function and impeding blood flow through the liver. Causes include malnutrition, inflammation (bacterial or viral), and poisons (e.g., alcohol, carbon tetrachloride, acetaminophen). Cirrhosis is the fourth leading cause of death in the United States among people ages 35 to 55 and represents a serious threat to long-term health.
CARE SETTING
May be hospitalized on a medical unit during initial or recurrent acute episodes with potentially life-threatening complications. Otherwise, this condition is handled at the community level.
RELATED CONCERNS
Alcohol: acute withdrawal
Substance dependence/abuse rehabilitation
Fluid and electrolyte imbalances
Psychosocial aspects of care
Renal dialysis
Renal failure: acute
Total nutritional support: parenteral/enteral feeding
Upper gastrointestinal/esophageal bleeding
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CARE SETTING
May be hospitalized on a medical unit during initial or recurrent acute episodes with potentially life-threatening complications. Otherwise, this condition is handled at the community level.
RELATED CONCERNS
Alcohol: acute withdrawal
Substance dependence/abuse rehabilitation
Fluid and electrolyte imbalances
Psychosocial aspects of care
Renal dialysis
Renal failure: acute
Total nutritional support: parenteral/enteral feeding
Upper gastrointestinal/esophageal bleeding
DOWNLOAD WHOLE ARTICLE
Diabetes Mellitus
* A group of metabolic diseases characterized by elevated levels of glucose in the blood resulting from defects in insulin secretion, insulin action, insulin receptors or any combination of conditions.
* A chronic disorder of impaired glucose metabolism, protein and fat metabolism
BASIC PATHOLOGY : Insulin problem (deficiency or impaired action)
* Insulin is a hormone secreted by the BETA cells of the pancreas
* Stimulus of insulin- HYPERGLYCEMIA
* Action of insulin: it promotes entry of Glucose into the body cells by binding to the insulin receptor in the cell membrane
Insulin Metabolic Functions:
* 1. Transports and metabolizes GLUCOSE
* 2. Promotes GLYCOGENESIS
* 3. Promotes GLYCOLYSIS
* 4. Enhances LIPOGENESIS
* 5. Accelerates PROTEIN SYNTHESIS
RISK FACTORS for Diabetes Mellitus
* 1. Family History of diabetes
* 2. Obesity
* 3. Race/Ethnicity
* 4. Age of more than 45
* 5. Previously unidentified IFG/IGT
* 6. Hypertension
* 7. Hyperlipidemia
* 8. History of Gestational Diabetes Mellitus
CLASSIFICATION OF DM
1. Type 1 DM
* Insulin dependent Diabetes Mellitus
2. Type 2 DM
* Non-insulin dependent Diabetes Mellitus
3. Gestational DM
* Diabetes Mellitus diagnosed during pregnancy
4. DM associated with other conditions or syndromes
Other types of DM
* 1. Impaired Glucose Tolerance
* 2. Impaired Fasting Glucose
* 3. Pre-diabetes
TYPE 1- Diabetes Mellitus
This type of DM is characterized by the destruction of the pancreatic beta cells
Etiology:
1. Genetic susceptibility- HLA DR3 and DR4
2. Autoimmune response
3. Toxins, unidentified viruses and environmental factors
PATHOPHYSIOLOGY
* Destruction of BETA cells--> decreased insulin production --> uncontrolled glucose production by the liver--> hyperglycemia --> signs and symptoms
CLASSIC P’s
* Polyuria
* Polydipsia
* Polyphagia
TYPE 2- Diabetes Mellitus
* A type of DM characterized by insulin resistance and impaired insulin production
Etiology:
1. Unknown
2. Probably genetic and obesity
PATHOPHYSIOLOGY
* Decreased sensitivity of insulin receptor to insulin --> less uptake of glucose --> HYPERGLYCEMIA
* Decreased insulin production --> diminished insulin action --> hyperglycemia --> signs and symptoms
* BUT (+) insulin in small amount --> prevent breakdown of fats --> DKA is unusual
GESTATIONAL Diabetes Mellitus
* Any degree of glucose intolerance with its onset during pregnancy
* Usually detected between 24-28th week gestation
* Blood glucose returns to normal after delivery of the infant
* NEVER administer ORAL HYPOGLYCEMIC AGENTS to PREGNANT MOTHERS!
ASSESSMENT FINDINGS
* 1. Classic 3 P’s
* 2. Fatigue
* 3. Body weakness
* 4. Visual changes
* 5. Slow wound healing
* 6. Recurrent skin and mucus membrane infections
DIAGNOSTIC TESTS
* 1. FBS- > 126
* 2. RBS- >200
* 3. OGTT- > 200
* 4. HgbA1- for monitoring!!
* 5. Urine glucose
* 6. Urine ketones
DIAGNOSTIC CRITERIA
* 1. FBS equal to or greater than 126 mg/dL (7.0mmol/L)
* (Normal 8 hour FBS- 80-109 mg/dL)
* 2. OGTT value 1 and 2 hours post-prandial equal to or greater than 200 mg/dL
* Normal OGTT 1 and 2 hours post-prandial- is
* 140 mg/dL
* 3. RBS of equal to or greater than 200 mg/dL PLUS the 3 P’s
NURSING MANAGEMENT OF DM
* The main goal is to NORMALIZE insulin activity and blood glucose level by:
Nutritional modification
2. Regular Exercise
3. Regular Glucose Monitoring
4. Drug therapy
5. Client Education
The Patient with DM
* HISTORY
* Symptoms and characteristics
* PHYSICAL EXAMINATION
* VS, BMI, Fundoscopy, and Neuro assessment
* LABORATORY EXAMINATION
* FBS, RBS, HgbA1c, lipid profile, ECG, and Urinalysis
* REFERRALS
* Ophthalmologist, Podiatrist, Dietician, etc..
DM Nutritional management
* 1.Review the patient’s diet history to identify eating habits and lifestyle
* 2. Coordinate with the dietician in meal planning for weight loss
* 3. Plan for the caloric intake distributed as follows- CHO 50-60%; Fats 20-30%; and Proteins 10-20%
* 4. Advise moderation in alcohol intake
* 5. Using artificial sweeteners is acceptable
DM Exercise management
* 1. Teach that exercise can lower the blood glucose level
* 2. Diabetics must first control the glucose level before initiating exercise programs.
* 3. Offer extra food /calories before engaging in exercise
* 4. Offer snacks at the end of the exercise period if patient is on insulin treatment.
* 5. Advise that exercise should be done at the same time every day, preferably when blood glucose levels are at their peak
* Regular exercise, not sporadic exercise, should be encouraged.
* 7. For most patient, WALKING is the safe and beneficial form of exercise
Glucose Self Monitoring
* Self-monitoring of blood glucose (SMBG) enables the patient to adjust the treatment regimen to obtain optimal glucose control
* Most common method involves obtaining a drop of capillary blood applied to a test strip.
* The usual recommended frequency is TWO-FOUR times a day.
When is it done?
* At the peak action time of the medication to evaluate the need for adjustments.
* To evaluate BASAL insulin --> test before meals
* To titrate bolus or regular and lispro--> test 2 hours after meals.
* To evaluate the glucose level of those taking ORAL hypoglycemics --> test before and two hours after meals.
Diabetes Mellitus Monitoring therapy
* Testing the glycosylated hemoglobin (HbA1c)
* This glycosylated hemoglobin refers to the blood test that reflects the average blood glucose over a period of TWO to THREE months.
* Normal value is 4 to 6 %
* No patient preparation is needed for this testing
* Done to monitor therapy
Diabetes Mellitus
* Urine testing for glucose
* Benedict’s test
* Urine testing for ketones
* Ketones are by-products of fat breakdown
* Urine testing for ketones
* This is performed whenever TYPE 1 DM have glucosuria or persistent elevation of blood glucose, during illness, and in gestational diabetes
DM Drug therapy
DRUG THERAPY and MANAGEMENT
* Usually, this type of management is employed if diet modification and exercise cannot control the blood glucose level.
* Because the patient with TYPE 1 DM cannot produce insulin, exogenous insulin must be administered for life.
* TYPE 2 DM may have decreased insulin production, ORAL agents that stimulate insulin production are usually employed.
PHARMACOLOGIC INSULIN
* This may be grouped into several categories according to:
1. Source- Human, pig, or cow
2. Onset of action- Rapid-acting, short-acting, intermediate-acting, long-acting and very long acting
* This may be grouped into several categories according to:
3. Pure or mixed concentration
4. Manufacturer of drug
GENERALITIES
* 1. Human insulin preparations have a shorter duration of action than animal source
* 2. Animal sources of insulin have animal proteins that may trigger allergic reaction and they may stimulate antibody production that may bind the insulin, slowing the action
* 3. ONLY Regular insulin can be used INTRAVENOUSLY!
* 4. Insulin are measured in INTERNATIONAL UNITS or “iu”
* 5. There is a specified insulin injection calibrated in units
RAPID ACTING INSULIN
* Lispro (Humalog) and Insulin Aspart (Novolog)
* Produces a more rapid effect and with a shorter duration than any other insulin preparation
* ONSET- 5-15 minutes
* PEAK- 1 hour
* DURATION- 3 hours
* Instruct patient to eat within 5 to 15 minutes after injection
REGULAR INSULIN
* Also called Short-acting insulin
* “R”
* Usually Clear solution administered 30 minutes before a meal
* ONSET- 30 minutes to 1 hour
* PEAK- 2 to 3 hours
* DURATION- 4 to 6 hours
INTERMEDIATE ACTING INSULIN
* Called “NPH” or “LENTE”
* Appears white and cloudy
* ONSET- 2-4 hours
* PEAK- 4 to 6-12 hours
* DURATION- 16-20 hours
LONG- ACTING INSULIN
* “UltraLENTE”
* Referred to as “peakless” insulin
* ONSET- 6-8 hours
* PEAK- 12-16 hours
* DURATION- 20-30 hours
HEALTH TEACHING
Regarding Insulin SELF- Administration
* 1. Insulin is administered at home subcutaneously
* 2. Cloudy insulin should be thoroughly mixed by gently inverting the vial or ROLLING between the hands
* 3. Insulin NOT IN USE should be stored in the refrigerator, BUT avoid freezing/extreme temperature
* 4. Insulin IN USE should be kept at room temperature to reduce local irritation at the injection site
* 5. INSULIN may be kept at room temperature up to 1 month
* 6. Select syringes that match the insulin concentration.
* U-100 means 100 units per mL
* Instruct the client to draw up the REGULAR (clear) Insulin FIRST before drawing the intermediate acting (cloudy) insulin
* 8. Pre-filled syringes can be prepared and should be kept in the refrigerator with the needle in the UPRIGHT position to avoid clogging the needle
* 9. The four main areas for insulin injection are- ABDOMEN, UPPER ARMS, THIGHS and HIPS
* Insulin is absorbed fastest in the abdomen and slowest in the hips
* Instruct the client to rotate the areas of injection, but exhaust all available sites in one area first before moving into another area.
* 10. Alcohol may not be used to cleanse the skin
* 11. Utilize the subcutaneous injection technique- commonly, a 45-90 degree angle.
* 12. No need to instruct for aspirating the needle
* 13. Properly discard the syringe after use.
T-I-E
Test blood--> Inject insulin --> Eat food
COMPLICATIONS OF INSULIN THERAPY
1. LOCAL ALLERGIC REACTIONS
* Redness, swelling, tenderness and induration appearing 1-2 hours after injection
* Usually occurs in the beginning stage of therapy
1. Local allergic reactions
* Disappears with continued use
* Antihistamine can be given 1 hour before injection time
* Porcine and bovine insulin preparations have a higher tendency to produce this reaction.
2. SYSTEMIC ALLERGIC REACTIONS
* Very rare
* Generalized urticaria is the manifestation
* Treatment is desensitization
3. INSULIN DYSTROPHY
* A localized reaction in the form of lipoatrophy or lipohypertrophy
* Lipoatrophy- loss of subcutaneous fat usually caused by the utilization of animal insulin
* Lipohypertrophy- development of fibrofatty masses, usually caused by repeated use of injection site
4. INSULIN RESISTANCE
* Most commonly caused by OBESITY
* Defined as daily insulin requirement of more than 200 units
* Management- Steroids and use of more concentrated insulin
5. MORNING HYPERGLYCEMIA
* Elevated blood sugar upon arising in the morning
* Caused by insufficient level of insulin
* DAWN phenomenon
* SOMOGYI effect
* INSULIN WANING
DAWN PHENOMENON
* Relatively normal blood glucose until about 3 am, when the glucose level begins to RISE
* Results from the nightly surges of GROWTH HORMONE secretion
* Management: Bedtime injection of NPH
SOMOGYI EFFECT
* Normal or elevated blood glucose at bedtime, decrease blood glucose at 2-3 am due to hypoglycemic levels and a subsequent increase in blood glucose (rebound hypergycemia)
* Nocturnal hypoglycemia followed by rebound hyperglycemia
* Due to the production of counter regulatory hormones- glucagon. cortisol and epinephrine
* Management- decrease evening dose of NPH or increase bedtime snack
INSULIN WANING
* Progressive rise in blood glucose from bedtime to morning
* Seen when the NPH evening dose is administered before dinner
* Management: Move the insulin injection to bedtime
ORAL HYPOGLYCEMIC AGENTS
* These may be effective when used in TYPE 2 DM that cannot be treated with diet and exercise
* These are NEVER used in pregnancy!
* There are several agents:
* Sulfonylureas
* Biguanides
* Alpha-glucosidase inhibitors
* Thiazolidinediones
* Meglitinides
SULFONYLUREAS
* MOA- stimulates the beta cells of the pancreas to secrete insulin
* Classified as to generations- first and second generations
* FIRST GENERATION- Acetoheximide, Chlorpropamide, Tolazamide and Tolbutamide
* SECOND GENERATION- Glipizide, Glyburide, Glibenclamide, Glimepiride
* The most common side –effects of these medications are Gastro-intestinal upset and dermatologic reactions.
* HYPOGLYCEMIA is also a very important side-effect
* Chlorpropamide has a very long duration of action. This also produces a disulfiram-like reaction when taken with alcohol
* Second generation drugs have shorter duration with metabolism in the kidney and liver and are the choice for elderly patients
BIGUANIDES- “ Formin”
* MOA- Facilitate the action of insulin on the peripheral receptors
* These can only be used in the presence of insulin
* They have no effect on the beta cells of the pancreas
* Metformin (Glucophage) and Phenformin are examples
* The most important side effect is LACTIC ACIDOSIS!
* These are not given to patient with renal impairment
* These drugs are usually given with a sulfonylurea to enhance the glucose-lowering effect more than the use of each drug individually
ALPHA-GLUCOSIDASE INHIBITORS
* MOA- Delay the absorption of glucose in the GIT
* Result is a lower post-prandial blood glucose level
* They do not affect insulin secretion or action!
* Side-effect: DIARRHEA and FLATULENCE
* Examples of AGI are Acarbose and Miglitol
* They are not absorbed systemically and are very safe
* They can be used alone or in combination with other OHA
* Side-effect if used with other drug is HYPOGLYCEMIA
* Note that sucrose absorption is impaired and IV glucose is the therapy for the hypoglycemia
THIAZOLIDINEDIONES
* MOA- Enhance insulin action at the receptor site
* They do not stimulate insulin secretion
* Examples- Rosiglitazone, Pioglitazone
* These drugs affect LIVER FUNCTION
* Can cause resumption of OVULATION in peri-menopausal anovulatory women
MEGLITINIDES
* MOA- Stimulate the secretion of insulin by the beta cells
* Examples- Repaglinide and Nateglinide
* They have a shorter duration and fast action
* Should be taken BEFORE meals to stimulate the release of insulin from the pancreas
* Principal side-effect of meglitinides- hypoglycemia
* Can be used alone or in combination
ACUTE COMPLICATIONS OF DM
* Hypoglycemia
* Diabetic ketoacidosis
* Hyperglycemic hyperosmolar non-ketotic syndrome (HHNS)
* Macrovascular complications- MI, Stroke, Atherosclerosis, CAD, and Peripheral vascular disease
* Microvascular complications- micro-angiopathy, retinopathy, nephropathy
* Peripheral neuropathy
HYPOGLYCEMIA
* Blood glucose level less than 50 to 60 mg/dL
* Causes: Too much insulin/OHA, too little food and excessive physical activity
* Mild- 40-60
* Moderate- 20-40
* Severe- less than 20
ASSESSMENT FINDINGS
* 1. Sympathetic manifestations- sweating, tremors, palpitations, nervousness, tachycardia and hunger
* 2. CNS manifestations- inability to concentrate, headache, lightheadedness, confusion, memory lapses, slurred speech, impaired coordination, behavioral changes, double vision and drowsiness
* DIAGNOSTIC FINDINGS
* RBS- less than 50-60 mg/dL level
Nursing Interventions
* 1. Immediate treatment with the use of foods with simple sugar- glucose tablets, fruit juice, table sugar, honey or hard candies
* 2. For unconscious patients- glucagon injection 1 mg IM/SQ; or IV 25 to 50 mL of D50/50
* 3. re-test glucose level in 15 minutes and re-treat if less than 75 mg/dL
* 4. Teach patient to refrain from eating high-calorie, high-fat desserts
* 5. Advise in-between snacks, especially when physical activity is increased
* 6. Teach the importance of compliance to medications
Diabetic Ketoacidosis
* This is cause by the absence of insulin leading to fat breakdown and production of ketone bodies
* Three main clinical features:
* 1. HYPERGLYCEMIA
* 2. DEHYDRATION & electrolyte loss
* 3. ACIDOSIS
PATHOPHYSIOLOGY
* No insulin--> reduced glucose breakdown and increased liver glucose production --> Hyperglycemia
* Hyperglycemia--> kidney attempts to excrete glucose --> increased osmotic load --> diuresis --> Dehydration
* No glucose in the cell--> fat is broken down for energy --> ketone bodies are produced--> Ketoacidosis
Risk factors
* 1. infection or illness- common
* 2. stress
* 3. undiagnosed DM
* 4. inadequate insulin, missed dose of insulin
ASSESSMENT FINDINGS
* 1. 3 P’s
* 2. Headache, blurred vision and weakness
* 3. Orthostatic hypotension
* 4. Nausea, vomiting and abdominal pain
* 5. Acetone (fruity) breath
* 6. Hyperventilation or KUSSMAUL’s breathing
LABORATORY FINDINGS
* 1. Blood glucose level of 300-800 mg/dL
* 2. Urinary ketones
* 3. ABG result of metabolic acidosis- LOW pH, LOW pCO2 as a compensation, LOW bicarbonate
* 4. Electrolyte imbalances- potassium levels may be HIGH due to acidosis and dehydration
NURSING INTERVENTIONS
* 1. Assist in the correction of dehydration
* Up to 6 liters of fluid may be ordered for infusion, initially NSS then D5W
* Monitor hydration status
* Monitor I and O
* Monitor for volume overload
* 2. Assist in restoring Electrolytes
* Kidney function is FIRST determined before giving potassium supplements!
* 3. Reverse the Acidosis
* REGULAR insulin injection is ordered IV bolus 5-10 units
* The insulin is followed by drip infusion in units per hour
* BICARBONATE is not used!
* A serious condition in which hyperosmolarity and extreme hyperglycemia predominate
* Ketosis is minimal
* Onset is slow and takes hours to days to develop
* Lack of insulin action or Insulin resistance --> hyperglycemia
* Hyperglycemia--> osmotic diuresis --> loss of water and electrolytes
HHNS
PATHOPHYSIOLOGY
* Insulin is too low to prevent hyperglycemia but enough to prevent fat breakdown
* Occurs most commonly in type 2 DM, ages 50-70
Precipitating factors
* 1. Infection
* 2. Stress
* 3. Surgery
* 4. Medication like thiazides
* 5. Treatment like dialysis
ASSESSMENT FINDINGS
* 1. Profound dehydration
* 2. Hypotension
* 3. Tachycardia
* 4. Altered sensorium
* 5. Seizures and hemiparesis
DIAGNOSTIC TESTS
* 1. Blood glucose- 600 to 1,200 mg/dL
* 2. Blood osmolality- 350 mOsm/L
* 3. Electrolyte abnormalities
NURSING INTERVENTIONS
* Approach is similar to the DKA
* 1. Correction of Dehydration by IVF
* 2. Correction of electrolyte imbalance by replacement therapy
* 3. Administration of insulin injection and drips
* 4. Continuous monitoring of urine output
MACROVASCULAR CX
Nursing management
* 1. Diet modification
* 2. Exercise
Nursing management
* 3. Prevention and treatment of underlying conditions such as MI, CAD and stroke
* 4. Administration of prescribed medications for hypertension, hyperlipidemia and obesity
* Retinopathy- a painless deterioration of the small blood vessels in the retina, may be classified as to background retinopathy, pre-proliferative and proliferative retinopathy
* Permanent vision changes and blindness can occur
Retinopathy-ASSESSMENT FINDINGS
* Blurry vision
* Spotty vision
* Asymptomatic
Retinopathy: Diagnostic findings
* 1. Fundoscopy
* 2. Fluorescein angiography
* Painless procedure
* Side-effects- discoloration of the skin and urine for 12 hours, some allergic reactions, nausea
* Flash of camera may be slightly uncomfortable
NURSING INTERVENTIONS
* 1. Assist in diagnostic procedure
* 2. Assist in the preparation for surgery- laser photocoagulation
* 3. Health teaching regarding prevention of retinopathy by regular ophthalmic examinations, good glucose control and self-management of eye care regimens
* 4. Maintain client safety
DIABETIC NEPHROPATHY
* Progressive deterioration of kidney function
DIABETIC NEPHROPATHY
* HYPERGLYCEMIA--> causes the kidney filtration mechanism to be stressed --> blood proteins leak into the urine
* Pressure in the kidney blood vessels increases--> stimulate the development of nephropathy
ASSESSMENT findings for diabetic nephropathy
* 1. Albuminuria
* 2. Anemia
* 3. Acidosis
* 4. Fluid volume overload
* 5. Oliguria
* 6. Hypertension
* 7. UTI
NURSING MANAGEMENT
1. Assist in the control of hypertension- use of ACE inhibitor
2. Provide a low sodium and low protein diet
3. Administer prescribed medication for UTI
4. Assist in dialysis
5. Prepare patient for renal transplantation, if indicated
Diabetic Neuropathy
* A group of disorders that affect all type of nerves including the peripheral, autonomic and spinal nerves
* Two most common types of Diabetic Neuropathy are sensori-motor polyneuropathy and autonomic neuropathy
ASSESSMENT findings
* 1. paresthesias- prickling, tingling or heightened sensation
* 2. decreased proprioception
* 3. decreased sensation of light touch
* 4. unsteady gait
* 5. decreased tendon reflexes
Nursing Management
* 1. Provide teaching that good glucose control is very important to prevent its development
* 2. Manage the pain by analgesics, antidepressants and nerve stimulation
Autonomic Neuropathy- ASSESSMENT findings
* 1. Silent, painless ischemia
* 2. delayed gastric emptying
* 3. orthostatic hypotension
* 4. N/V and bloating sensation
* 5. urinary retention
* 6. sexual dysfunction
Autonomic Neuropathy-Nursing management
* 1. Educate about the avoidance of strenuous physical activity
* 2. Stress the importance of good glucose control to delay the development
* 3. Provide LOW-fat, small frequent feedings
* 4. Administer bulk-forming laxatives for diabetic diarrhea
* 5. Provide HIGH-fiber diet for diabetic constipation
RISK FACTORS for the development of foot and leg ulcers
* 1. More than 10 years diabetic
* 2. Age of more than 40
* 3. Smoking
* 4. Anatomic deformities
* 5. History of previous leg ulcers or amputation
MANAGEMENT of Foot Ulcers
* Teach patient proper care of the foot
* Daily assessment of the foot
* Use of mirror to inspect the bottom
* Inspect the surface of shoes for any rough spots or foreign objects
* Properly dry the feet
* Instruct to wear closed-toe shoes that fit well, recommend use of low-heeled shoes
* Instruct patient NEVER to walk barefoot, never to use heating pads, open-toed shoes and soaking feet
* Trim toenails STRAIGHT ACROSS and file sharp corners
* Instruct to avoid smoking and over-the counter medications and home remedies for foot problems
INSULIN
Types of Insulin
1. Rapid Acting Insulin- Ex. Insulin Lispro ( Humalog)
2. Short Acting Insulin- Ex. Regular Insulin (Humulin R)
3. Intermediate Acting Insulin Ex. NPH Insulin ( Humulin N)
4. Long Acting Insulin- Ex. Insulin Glergene ( Lantus), InsulinDetermir
( Levemir)
5. Fixed Combination of N and R- Ex. 70/30%- 70% N, 30 % R
6. Fixed Combination- Ex. Humalog Mix 75/25, 75% lispro protamine,
25% lispro insulin
Insulin
> Enhances transmembrane passage of glucose across cell membrane.
> Promotes conversion of glucose to glycogen.
Common Uses
1. Oral hypopglycemic Agents- Type 2 DM
2. Insulin- Type 1 DM
Common adverse effects: hypoglycemia.
Types
1. Rapid- acting insulin
> Onset: within 15 minutes
> Peak: 30-90 minutes
> Duration: 3-4 hrs
2. Short- acting insulin
> Onset: 30-60 minutes
> Peak: 2-3 hrs
> Duration: 3-6 hrs
3. Intermediate –acting insulin
> Onset: 2-4 hrs
> Peak: 6-10 hrs
> Duration: 10-16 hrs
4. Long-acting insulin
> Onset: 2 hrs
> Peakless
> Duration: 24 hrs
5. Fixed Combination of N and R
> Onset: 30-60 minutes
> Peak: 6-10hrs
> Duration: 10-16 hrs
6. Fixed combination
> Onset: 15 minutes
> Peak: 1-6.5 hrs (average 2.5 hrs)
> Duration: 10-16 hrs
* A chronic disorder of impaired glucose metabolism, protein and fat metabolism
BASIC PATHOLOGY : Insulin problem (deficiency or impaired action)
* Insulin is a hormone secreted by the BETA cells of the pancreas
* Stimulus of insulin- HYPERGLYCEMIA
* Action of insulin: it promotes entry of Glucose into the body cells by binding to the insulin receptor in the cell membrane
Insulin Metabolic Functions:
* 1. Transports and metabolizes GLUCOSE
* 2. Promotes GLYCOGENESIS
* 3. Promotes GLYCOLYSIS
* 4. Enhances LIPOGENESIS
* 5. Accelerates PROTEIN SYNTHESIS
RISK FACTORS for Diabetes Mellitus
* 1. Family History of diabetes
* 2. Obesity
* 3. Race/Ethnicity
* 4. Age of more than 45
* 5. Previously unidentified IFG/IGT
* 6. Hypertension
* 7. Hyperlipidemia
* 8. History of Gestational Diabetes Mellitus
CLASSIFICATION OF DM
1. Type 1 DM
* Insulin dependent Diabetes Mellitus
2. Type 2 DM
* Non-insulin dependent Diabetes Mellitus
3. Gestational DM
* Diabetes Mellitus diagnosed during pregnancy
4. DM associated with other conditions or syndromes
Other types of DM
* 1. Impaired Glucose Tolerance
* 2. Impaired Fasting Glucose
* 3. Pre-diabetes
TYPE 1- Diabetes Mellitus
This type of DM is characterized by the destruction of the pancreatic beta cells
Etiology:
1. Genetic susceptibility- HLA DR3 and DR4
2. Autoimmune response
3. Toxins, unidentified viruses and environmental factors
PATHOPHYSIOLOGY
* Destruction of BETA cells--> decreased insulin production --> uncontrolled glucose production by the liver--> hyperglycemia --> signs and symptoms
CLASSIC P’s
* Polyuria
* Polydipsia
* Polyphagia
TYPE 2- Diabetes Mellitus
* A type of DM characterized by insulin resistance and impaired insulin production
Etiology:
1. Unknown
2. Probably genetic and obesity
PATHOPHYSIOLOGY
* Decreased sensitivity of insulin receptor to insulin --> less uptake of glucose --> HYPERGLYCEMIA
* Decreased insulin production --> diminished insulin action --> hyperglycemia --> signs and symptoms
* BUT (+) insulin in small amount --> prevent breakdown of fats --> DKA is unusual
GESTATIONAL Diabetes Mellitus
* Any degree of glucose intolerance with its onset during pregnancy
* Usually detected between 24-28th week gestation
* Blood glucose returns to normal after delivery of the infant
* NEVER administer ORAL HYPOGLYCEMIC AGENTS to PREGNANT MOTHERS!
ASSESSMENT FINDINGS
* 1. Classic 3 P’s
* 2. Fatigue
* 3. Body weakness
* 4. Visual changes
* 5. Slow wound healing
* 6. Recurrent skin and mucus membrane infections
DIAGNOSTIC TESTS
* 1. FBS- > 126
* 2. RBS- >200
* 3. OGTT- > 200
* 4. HgbA1- for monitoring!!
* 5. Urine glucose
* 6. Urine ketones
DIAGNOSTIC CRITERIA
* 1. FBS equal to or greater than 126 mg/dL (7.0mmol/L)
* (Normal 8 hour FBS- 80-109 mg/dL)
* 2. OGTT value 1 and 2 hours post-prandial equal to or greater than 200 mg/dL
* Normal OGTT 1 and 2 hours post-prandial- is
* 140 mg/dL
* 3. RBS of equal to or greater than 200 mg/dL PLUS the 3 P’s
NURSING MANAGEMENT OF DM
* The main goal is to NORMALIZE insulin activity and blood glucose level by:
Nutritional modification
2. Regular Exercise
3. Regular Glucose Monitoring
4. Drug therapy
5. Client Education
The Patient with DM
* HISTORY
* Symptoms and characteristics
* PHYSICAL EXAMINATION
* VS, BMI, Fundoscopy, and Neuro assessment
* LABORATORY EXAMINATION
* FBS, RBS, HgbA1c, lipid profile, ECG, and Urinalysis
* REFERRALS
* Ophthalmologist, Podiatrist, Dietician, etc..
DM Nutritional management
* 1.Review the patient’s diet history to identify eating habits and lifestyle
* 2. Coordinate with the dietician in meal planning for weight loss
* 3. Plan for the caloric intake distributed as follows- CHO 50-60%; Fats 20-30%; and Proteins 10-20%
* 4. Advise moderation in alcohol intake
* 5. Using artificial sweeteners is acceptable
DM Exercise management
* 1. Teach that exercise can lower the blood glucose level
* 2. Diabetics must first control the glucose level before initiating exercise programs.
* 3. Offer extra food /calories before engaging in exercise
* 4. Offer snacks at the end of the exercise period if patient is on insulin treatment.
* 5. Advise that exercise should be done at the same time every day, preferably when blood glucose levels are at their peak
* Regular exercise, not sporadic exercise, should be encouraged.
* 7. For most patient, WALKING is the safe and beneficial form of exercise
Glucose Self Monitoring
* Self-monitoring of blood glucose (SMBG) enables the patient to adjust the treatment regimen to obtain optimal glucose control
* Most common method involves obtaining a drop of capillary blood applied to a test strip.
* The usual recommended frequency is TWO-FOUR times a day.
When is it done?
* At the peak action time of the medication to evaluate the need for adjustments.
* To evaluate BASAL insulin --> test before meals
* To titrate bolus or regular and lispro--> test 2 hours after meals.
* To evaluate the glucose level of those taking ORAL hypoglycemics --> test before and two hours after meals.
Diabetes Mellitus Monitoring therapy
* Testing the glycosylated hemoglobin (HbA1c)
* This glycosylated hemoglobin refers to the blood test that reflects the average blood glucose over a period of TWO to THREE months.
* Normal value is 4 to 6 %
* No patient preparation is needed for this testing
* Done to monitor therapy
Diabetes Mellitus
* Urine testing for glucose
* Benedict’s test
* Urine testing for ketones
* Ketones are by-products of fat breakdown
* Urine testing for ketones
* This is performed whenever TYPE 1 DM have glucosuria or persistent elevation of blood glucose, during illness, and in gestational diabetes
DM Drug therapy
DRUG THERAPY and MANAGEMENT
* Usually, this type of management is employed if diet modification and exercise cannot control the blood glucose level.
* Because the patient with TYPE 1 DM cannot produce insulin, exogenous insulin must be administered for life.
* TYPE 2 DM may have decreased insulin production, ORAL agents that stimulate insulin production are usually employed.
PHARMACOLOGIC INSULIN
* This may be grouped into several categories according to:
1. Source- Human, pig, or cow
2. Onset of action- Rapid-acting, short-acting, intermediate-acting, long-acting and very long acting
* This may be grouped into several categories according to:
3. Pure or mixed concentration
4. Manufacturer of drug
GENERALITIES
* 1. Human insulin preparations have a shorter duration of action than animal source
* 2. Animal sources of insulin have animal proteins that may trigger allergic reaction and they may stimulate antibody production that may bind the insulin, slowing the action
* 3. ONLY Regular insulin can be used INTRAVENOUSLY!
* 4. Insulin are measured in INTERNATIONAL UNITS or “iu”
* 5. There is a specified insulin injection calibrated in units
RAPID ACTING INSULIN
* Lispro (Humalog) and Insulin Aspart (Novolog)
* Produces a more rapid effect and with a shorter duration than any other insulin preparation
* ONSET- 5-15 minutes
* PEAK- 1 hour
* DURATION- 3 hours
* Instruct patient to eat within 5 to 15 minutes after injection
REGULAR INSULIN
* Also called Short-acting insulin
* “R”
* Usually Clear solution administered 30 minutes before a meal
* ONSET- 30 minutes to 1 hour
* PEAK- 2 to 3 hours
* DURATION- 4 to 6 hours
INTERMEDIATE ACTING INSULIN
* Called “NPH” or “LENTE”
* Appears white and cloudy
* ONSET- 2-4 hours
* PEAK- 4 to 6-12 hours
* DURATION- 16-20 hours
LONG- ACTING INSULIN
* “UltraLENTE”
* Referred to as “peakless” insulin
* ONSET- 6-8 hours
* PEAK- 12-16 hours
* DURATION- 20-30 hours
HEALTH TEACHING
Regarding Insulin SELF- Administration
* 1. Insulin is administered at home subcutaneously
* 2. Cloudy insulin should be thoroughly mixed by gently inverting the vial or ROLLING between the hands
* 3. Insulin NOT IN USE should be stored in the refrigerator, BUT avoid freezing/extreme temperature
* 4. Insulin IN USE should be kept at room temperature to reduce local irritation at the injection site
* 5. INSULIN may be kept at room temperature up to 1 month
* 6. Select syringes that match the insulin concentration.
* U-100 means 100 units per mL
* Instruct the client to draw up the REGULAR (clear) Insulin FIRST before drawing the intermediate acting (cloudy) insulin
* 8. Pre-filled syringes can be prepared and should be kept in the refrigerator with the needle in the UPRIGHT position to avoid clogging the needle
* 9. The four main areas for insulin injection are- ABDOMEN, UPPER ARMS, THIGHS and HIPS
* Insulin is absorbed fastest in the abdomen and slowest in the hips
* Instruct the client to rotate the areas of injection, but exhaust all available sites in one area first before moving into another area.
* 10. Alcohol may not be used to cleanse the skin
* 11. Utilize the subcutaneous injection technique- commonly, a 45-90 degree angle.
* 12. No need to instruct for aspirating the needle
* 13. Properly discard the syringe after use.
T-I-E
Test blood--> Inject insulin --> Eat food
COMPLICATIONS OF INSULIN THERAPY
1. LOCAL ALLERGIC REACTIONS
* Redness, swelling, tenderness and induration appearing 1-2 hours after injection
* Usually occurs in the beginning stage of therapy
1. Local allergic reactions
* Disappears with continued use
* Antihistamine can be given 1 hour before injection time
* Porcine and bovine insulin preparations have a higher tendency to produce this reaction.
2. SYSTEMIC ALLERGIC REACTIONS
* Very rare
* Generalized urticaria is the manifestation
* Treatment is desensitization
3. INSULIN DYSTROPHY
* A localized reaction in the form of lipoatrophy or lipohypertrophy
* Lipoatrophy- loss of subcutaneous fat usually caused by the utilization of animal insulin
* Lipohypertrophy- development of fibrofatty masses, usually caused by repeated use of injection site
4. INSULIN RESISTANCE
* Most commonly caused by OBESITY
* Defined as daily insulin requirement of more than 200 units
* Management- Steroids and use of more concentrated insulin
5. MORNING HYPERGLYCEMIA
* Elevated blood sugar upon arising in the morning
* Caused by insufficient level of insulin
* DAWN phenomenon
* SOMOGYI effect
* INSULIN WANING
DAWN PHENOMENON
* Relatively normal blood glucose until about 3 am, when the glucose level begins to RISE
* Results from the nightly surges of GROWTH HORMONE secretion
* Management: Bedtime injection of NPH
SOMOGYI EFFECT
* Normal or elevated blood glucose at bedtime, decrease blood glucose at 2-3 am due to hypoglycemic levels and a subsequent increase in blood glucose (rebound hypergycemia)
* Nocturnal hypoglycemia followed by rebound hyperglycemia
* Due to the production of counter regulatory hormones- glucagon. cortisol and epinephrine
* Management- decrease evening dose of NPH or increase bedtime snack
INSULIN WANING
* Progressive rise in blood glucose from bedtime to morning
* Seen when the NPH evening dose is administered before dinner
* Management: Move the insulin injection to bedtime
ORAL HYPOGLYCEMIC AGENTS
* These may be effective when used in TYPE 2 DM that cannot be treated with diet and exercise
* These are NEVER used in pregnancy!
* There are several agents:
* Sulfonylureas
* Biguanides
* Alpha-glucosidase inhibitors
* Thiazolidinediones
* Meglitinides
SULFONYLUREAS
* MOA- stimulates the beta cells of the pancreas to secrete insulin
* Classified as to generations- first and second generations
* FIRST GENERATION- Acetoheximide, Chlorpropamide, Tolazamide and Tolbutamide
* SECOND GENERATION- Glipizide, Glyburide, Glibenclamide, Glimepiride
* The most common side –effects of these medications are Gastro-intestinal upset and dermatologic reactions.
* HYPOGLYCEMIA is also a very important side-effect
* Chlorpropamide has a very long duration of action. This also produces a disulfiram-like reaction when taken with alcohol
* Second generation drugs have shorter duration with metabolism in the kidney and liver and are the choice for elderly patients
BIGUANIDES- “ Formin”
* MOA- Facilitate the action of insulin on the peripheral receptors
* These can only be used in the presence of insulin
* They have no effect on the beta cells of the pancreas
* Metformin (Glucophage) and Phenformin are examples
* The most important side effect is LACTIC ACIDOSIS!
* These are not given to patient with renal impairment
* These drugs are usually given with a sulfonylurea to enhance the glucose-lowering effect more than the use of each drug individually
ALPHA-GLUCOSIDASE INHIBITORS
* MOA- Delay the absorption of glucose in the GIT
* Result is a lower post-prandial blood glucose level
* They do not affect insulin secretion or action!
* Side-effect: DIARRHEA and FLATULENCE
* Examples of AGI are Acarbose and Miglitol
* They are not absorbed systemically and are very safe
* They can be used alone or in combination with other OHA
* Side-effect if used with other drug is HYPOGLYCEMIA
* Note that sucrose absorption is impaired and IV glucose is the therapy for the hypoglycemia
THIAZOLIDINEDIONES
* MOA- Enhance insulin action at the receptor site
* They do not stimulate insulin secretion
* Examples- Rosiglitazone, Pioglitazone
* These drugs affect LIVER FUNCTION
* Can cause resumption of OVULATION in peri-menopausal anovulatory women
MEGLITINIDES
* MOA- Stimulate the secretion of insulin by the beta cells
* Examples- Repaglinide and Nateglinide
* They have a shorter duration and fast action
* Should be taken BEFORE meals to stimulate the release of insulin from the pancreas
* Principal side-effect of meglitinides- hypoglycemia
* Can be used alone or in combination
ACUTE COMPLICATIONS OF DM
* Hypoglycemia
* Diabetic ketoacidosis
* Hyperglycemic hyperosmolar non-ketotic syndrome (HHNS)
* Macrovascular complications- MI, Stroke, Atherosclerosis, CAD, and Peripheral vascular disease
* Microvascular complications- micro-angiopathy, retinopathy, nephropathy
* Peripheral neuropathy
HYPOGLYCEMIA
* Blood glucose level less than 50 to 60 mg/dL
* Causes: Too much insulin/OHA, too little food and excessive physical activity
* Mild- 40-60
* Moderate- 20-40
* Severe- less than 20
ASSESSMENT FINDINGS
* 1. Sympathetic manifestations- sweating, tremors, palpitations, nervousness, tachycardia and hunger
* 2. CNS manifestations- inability to concentrate, headache, lightheadedness, confusion, memory lapses, slurred speech, impaired coordination, behavioral changes, double vision and drowsiness
* DIAGNOSTIC FINDINGS
* RBS- less than 50-60 mg/dL level
Nursing Interventions
* 1. Immediate treatment with the use of foods with simple sugar- glucose tablets, fruit juice, table sugar, honey or hard candies
* 2. For unconscious patients- glucagon injection 1 mg IM/SQ; or IV 25 to 50 mL of D50/50
* 3. re-test glucose level in 15 minutes and re-treat if less than 75 mg/dL
* 4. Teach patient to refrain from eating high-calorie, high-fat desserts
* 5. Advise in-between snacks, especially when physical activity is increased
* 6. Teach the importance of compliance to medications
Diabetic Ketoacidosis
* This is cause by the absence of insulin leading to fat breakdown and production of ketone bodies
* Three main clinical features:
* 1. HYPERGLYCEMIA
* 2. DEHYDRATION & electrolyte loss
* 3. ACIDOSIS
PATHOPHYSIOLOGY
* No insulin--> reduced glucose breakdown and increased liver glucose production --> Hyperglycemia
* Hyperglycemia--> kidney attempts to excrete glucose --> increased osmotic load --> diuresis --> Dehydration
* No glucose in the cell--> fat is broken down for energy --> ketone bodies are produced--> Ketoacidosis
Risk factors
* 1. infection or illness- common
* 2. stress
* 3. undiagnosed DM
* 4. inadequate insulin, missed dose of insulin
ASSESSMENT FINDINGS
* 1. 3 P’s
* 2. Headache, blurred vision and weakness
* 3. Orthostatic hypotension
* 4. Nausea, vomiting and abdominal pain
* 5. Acetone (fruity) breath
* 6. Hyperventilation or KUSSMAUL’s breathing
LABORATORY FINDINGS
* 1. Blood glucose level of 300-800 mg/dL
* 2. Urinary ketones
* 3. ABG result of metabolic acidosis- LOW pH, LOW pCO2 as a compensation, LOW bicarbonate
* 4. Electrolyte imbalances- potassium levels may be HIGH due to acidosis and dehydration
NURSING INTERVENTIONS
* 1. Assist in the correction of dehydration
* Up to 6 liters of fluid may be ordered for infusion, initially NSS then D5W
* Monitor hydration status
* Monitor I and O
* Monitor for volume overload
* 2. Assist in restoring Electrolytes
* Kidney function is FIRST determined before giving potassium supplements!
* 3. Reverse the Acidosis
* REGULAR insulin injection is ordered IV bolus 5-10 units
* The insulin is followed by drip infusion in units per hour
* BICARBONATE is not used!
* A serious condition in which hyperosmolarity and extreme hyperglycemia predominate
* Ketosis is minimal
* Onset is slow and takes hours to days to develop
* Lack of insulin action or Insulin resistance --> hyperglycemia
* Hyperglycemia--> osmotic diuresis --> loss of water and electrolytes
HHNS
PATHOPHYSIOLOGY
* Insulin is too low to prevent hyperglycemia but enough to prevent fat breakdown
* Occurs most commonly in type 2 DM, ages 50-70
Precipitating factors
* 1. Infection
* 2. Stress
* 3. Surgery
* 4. Medication like thiazides
* 5. Treatment like dialysis
ASSESSMENT FINDINGS
* 1. Profound dehydration
* 2. Hypotension
* 3. Tachycardia
* 4. Altered sensorium
* 5. Seizures and hemiparesis
DIAGNOSTIC TESTS
* 1. Blood glucose- 600 to 1,200 mg/dL
* 2. Blood osmolality- 350 mOsm/L
* 3. Electrolyte abnormalities
NURSING INTERVENTIONS
* Approach is similar to the DKA
* 1. Correction of Dehydration by IVF
* 2. Correction of electrolyte imbalance by replacement therapy
* 3. Administration of insulin injection and drips
* 4. Continuous monitoring of urine output
MACROVASCULAR CX
Nursing management
* 1. Diet modification
* 2. Exercise
Nursing management
* 3. Prevention and treatment of underlying conditions such as MI, CAD and stroke
* 4. Administration of prescribed medications for hypertension, hyperlipidemia and obesity
* Retinopathy- a painless deterioration of the small blood vessels in the retina, may be classified as to background retinopathy, pre-proliferative and proliferative retinopathy
* Permanent vision changes and blindness can occur
Retinopathy-ASSESSMENT FINDINGS
* Blurry vision
* Spotty vision
* Asymptomatic
Retinopathy: Diagnostic findings
* 1. Fundoscopy
* 2. Fluorescein angiography
* Painless procedure
* Side-effects- discoloration of the skin and urine for 12 hours, some allergic reactions, nausea
* Flash of camera may be slightly uncomfortable
NURSING INTERVENTIONS
* 1. Assist in diagnostic procedure
* 2. Assist in the preparation for surgery- laser photocoagulation
* 3. Health teaching regarding prevention of retinopathy by regular ophthalmic examinations, good glucose control and self-management of eye care regimens
* 4. Maintain client safety
DIABETIC NEPHROPATHY
* Progressive deterioration of kidney function
DIABETIC NEPHROPATHY
* HYPERGLYCEMIA--> causes the kidney filtration mechanism to be stressed --> blood proteins leak into the urine
* Pressure in the kidney blood vessels increases--> stimulate the development of nephropathy
ASSESSMENT findings for diabetic nephropathy
* 1. Albuminuria
* 2. Anemia
* 3. Acidosis
* 4. Fluid volume overload
* 5. Oliguria
* 6. Hypertension
* 7. UTI
NURSING MANAGEMENT
1. Assist in the control of hypertension- use of ACE inhibitor
2. Provide a low sodium and low protein diet
3. Administer prescribed medication for UTI
4. Assist in dialysis
5. Prepare patient for renal transplantation, if indicated
Diabetic Neuropathy
* A group of disorders that affect all type of nerves including the peripheral, autonomic and spinal nerves
* Two most common types of Diabetic Neuropathy are sensori-motor polyneuropathy and autonomic neuropathy
ASSESSMENT findings
* 1. paresthesias- prickling, tingling or heightened sensation
* 2. decreased proprioception
* 3. decreased sensation of light touch
* 4. unsteady gait
* 5. decreased tendon reflexes
Nursing Management
* 1. Provide teaching that good glucose control is very important to prevent its development
* 2. Manage the pain by analgesics, antidepressants and nerve stimulation
Autonomic Neuropathy- ASSESSMENT findings
* 1. Silent, painless ischemia
* 2. delayed gastric emptying
* 3. orthostatic hypotension
* 4. N/V and bloating sensation
* 5. urinary retention
* 6. sexual dysfunction
Autonomic Neuropathy-Nursing management
* 1. Educate about the avoidance of strenuous physical activity
* 2. Stress the importance of good glucose control to delay the development
* 3. Provide LOW-fat, small frequent feedings
* 4. Administer bulk-forming laxatives for diabetic diarrhea
* 5. Provide HIGH-fiber diet for diabetic constipation
RISK FACTORS for the development of foot and leg ulcers
* 1. More than 10 years diabetic
* 2. Age of more than 40
* 3. Smoking
* 4. Anatomic deformities
* 5. History of previous leg ulcers or amputation
MANAGEMENT of Foot Ulcers
* Teach patient proper care of the foot
* Daily assessment of the foot
* Use of mirror to inspect the bottom
* Inspect the surface of shoes for any rough spots or foreign objects
* Properly dry the feet
* Instruct to wear closed-toe shoes that fit well, recommend use of low-heeled shoes
* Instruct patient NEVER to walk barefoot, never to use heating pads, open-toed shoes and soaking feet
* Trim toenails STRAIGHT ACROSS and file sharp corners
* Instruct to avoid smoking and over-the counter medications and home remedies for foot problems
INSULIN
Types of Insulin
1. Rapid Acting Insulin- Ex. Insulin Lispro ( Humalog)
2. Short Acting Insulin- Ex. Regular Insulin (Humulin R)
3. Intermediate Acting Insulin Ex. NPH Insulin ( Humulin N)
4. Long Acting Insulin- Ex. Insulin Glergene ( Lantus), InsulinDetermir
( Levemir)
5. Fixed Combination of N and R- Ex. 70/30%- 70% N, 30 % R
6. Fixed Combination- Ex. Humalog Mix 75/25, 75% lispro protamine,
25% lispro insulin
Insulin
> Enhances transmembrane passage of glucose across cell membrane.
> Promotes conversion of glucose to glycogen.
Common Uses
1. Oral hypopglycemic Agents- Type 2 DM
2. Insulin- Type 1 DM
Common adverse effects: hypoglycemia.
Types
1. Rapid- acting insulin
> Onset: within 15 minutes
> Peak: 30-90 minutes
> Duration: 3-4 hrs
2. Short- acting insulin
> Onset: 30-60 minutes
> Peak: 2-3 hrs
> Duration: 3-6 hrs
3. Intermediate –acting insulin
> Onset: 2-4 hrs
> Peak: 6-10 hrs
> Duration: 10-16 hrs
4. Long-acting insulin
> Onset: 2 hrs
> Peakless
> Duration: 24 hrs
5. Fixed Combination of N and R
> Onset: 30-60 minutes
> Peak: 6-10hrs
> Duration: 10-16 hrs
6. Fixed combination
> Onset: 15 minutes
> Peak: 1-6.5 hrs (average 2.5 hrs)
> Duration: 10-16 hrs
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